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  1. CFTR potentiators partially restore channel function to A561E‐CFTR, a cystic fibrosis mutant with a similar mechanism of dysfunction as F508del‐CFTR
  2. Revertant mutants modify, but do not rescue, the gating defect of the cystic fibrosis mutant G551D‐CFTR
  3. Loop diuretics are open-channel blockers of the cystic fibrosis transmembrane conductance regulator with distinct kinetics
  4. Acute inhibition of the cystic fibrosis transmembrane conductance regulator (CFTR) Cl- channel by thyroid hormones involves multiple mechanisms
  5. Impact of the cystic fibrosis mutation F508del-CFTR on renal cyst formation and growth