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  1. High-fat diet impairs glucose homeostasis by increased p16 beta-cell expression and alters glucose homeostasis of the progeny in a parental-sex dependent manner
  2. NGF and Its Role in Immunoendocrine Communication during Metabolic Syndrome
  3. Early Effects of Metabolic Syndrome on ATP-Sensitive Potassium Channels from Rat Pancreatic Beta Cells
  4. Molecular Insulin Actions Are Sexually Dimorphic in Lipid Metabolism
  5. Evidencias de transmisión intergeneracional de la obesidad y sus comorbilidades
  6. Sexual dimorphism in insulin resistance in a metabolic syndrome rat model
  7. Rat Pancreatic Beta-Cell Culture
  8. Ionic channels and insulin secretion in pancreatic beta cells
  9. Changes of maturing pancreatic beta cells
  10. Pancreatic β Cells in Metabolic Syndrome
  11. Metabolic Syndrome and Ionic Channels in Pancreatic Beta Cells
  12. Pancreatic Beta Cells in Metabolic Syndrome
  13. Differential expression of NGF and BDNF in rat adipose depots during early development and adulthood
  14. Metabolic syndrome induces changes in KATP-channels and calcium currents in pancreatic β-cells
  15. Early endocrine and molecular changes in metabolic syndrome models
  16. Changes in Insulin Secretion, KATP and Ba Currents in Rat Pancreatic Beta Cells from Rats with Metabolic Syndrome, Induced by a High Sucrose Diet