All Stories

  1. Unraveling the mechanisms of cardiolipin function: The role of oxidative polymerization of unsaturated acyl chains
  2. Mitochondrial Volume Regulation and Swelling Mechanisms in Cardiomyocytes
  3. Amyloid β induces cardiac dysfunction and neuro-signaling impairment in the heart of an Alzheimer’s disease model
  4. The Effects of Amyloid-β on Metabolomic Profiles of Cardiomyocytes and Coronary Endothelial Cells
  5. Crosstalk between adenine nucleotide transporter and mitochondrial swelling: experimental and computational approaches
  6. Analysis of Mitochondrial Function, Structure, and Intracellular Organization In Situ in Cardiomyocytes and Skeletal Muscles
  7. Mitochondria and ferroptosis
  8. Effects of Ferroptosis on the Metabolome in Cardiac Cells: The Role of Glutaminolysis
  9. Beta-Amyloid Instigates Dysfunction of Mitochondria in Cardiac Cells
  10. Analysis of Mitochondrial Calcium Retention Capacity in Cultured Cells: Permeabilized Cells Versus Isolated Mitochondria
  11. Dissecting the Crosstalk between Endothelial Mitochondrial Damage, Vascular Inflammation, and Neurodegeneration in Cerebral Amyloid Angiopathy and Alzheimer’s Disease
  12. Elucidating the contribution of mitochondrial glutathione to ferroptosis in cardiomyocytes
  13. Structural and functional remodeling of mitochondria as an adaptive response to energy deprivation
  14. The Role of Cytoskeletal Proteins in the Regulation of Mitochondrial Function
  15. Mitochondrial Involvement in the Molecular Mechanisms of Ischemia-Reperfusion Injury in the Heart
  16. Direct Mapping of Phospholipid Ferroptotic Death Signals in Cells and Tissues by Gas Cluster Ion Beam Secondary Ion Mass Spectrometry (GCIB‐SIMS)
  17. Mitochondrial respiratory supercomplexes in mammalian cells: structural versus functional role
  18. Association Between L-OPA1 Cleavage and Cardiac Dysfunction During Ischemia-Reperfusion Injury in Rats
  19. Mitochondria in Health and Diseases
  20. Mitochondria in Health and Diseases
  21. OPA1 regulates respiratory supercomplexes assembly: The role of mitochondrial swelling
  22. Cardiac Function is not Susceptible to Moderate Disassembly of Mitochondrial Respiratory Supercomplexes
  23. Crosstalk between Mitochondria and Cytoskeleton in Cardiac Cells
  24. In silico simulation of reversible and irreversible swelling of mitochondria: The role of membrane rigidity
  25. The Role of Adenine Nucleotide Translocase in the Assembly of Respiratory Supercomplexes in Cardiac Cells
  26. The Role of Mitochondria in the Mechanisms of Cardiac Ischemia-Reperfusion Injury
  27. Mitochondrial angiotensin receptors and cardioprotective pathways
  28. Elucidating the contribution of ETC complexes I and II to the respirasome formation in cardiac mitochondria
  29. Elucidating the Beneficial Role of PPAR Agonists in Cardiac Diseases
  30. Acetylation of Mitochondrial Proteins in the Heart: The Role of SIRT3
  31. Abstract 412: The Role of ETC Complexes I and II in the Respirasome Assembly in Heart Mitochondria
  32. Critical role of angiotensin II type 2 receptors in the control of mitochondrial and cardiac function in angiotensin II-preconditioned rat hearts
  33. The Effects of PPAR Stimulation on Cardiac Metabolic Pathways in Barth Syndrome Mice
  34. Computational Modeling of In Vitro Swelling of Mitochondria: A Biophysical Approach
  35. Current Challenges in Elucidating Respiratory Supercomplexes in Mitochondria: Methodological Obstacles
  36. Biophysical modeling of mitochondrial swelling
  37. Divergent Effects of Cyclophilin-D Inhibition on the Female Rat Heart: Acute Versus Chronic Post-Myocardial Infarction
  38. Angiotensin II-preconditioning is associated with increased PKCε/PKCδ ratio and prosurvival kinases in mitochondria
  39. Association between ROS production, swelling and the respirasome integrity in cardiac mitochondria
  40. Different approaches to modeling analysis of mitochondrial swelling
  41. Elucidating Mitochondrial Electron Transport Chain Supercomplexes in the Heart During Ischemia–Reperfusion
  42. Synchronism in mitochondrial ROS flashes, membrane depolarization and calcium sparks in human carcinoma cells
  43. High Sensitivity of SIRT3 Deficient Hearts to Ischemia-Reperfusion Is Associated with Mitochondrial Abnormalities
  44. Mitochondrial permeability transition in cardiac ischemia–reperfusion: whether cyclophilin D is a viable target for cardioprotection?
  45. The PPAR pan-agonist bezafibrate ameliorates cardiomyopathy in a mouse model of Barth syndrome
  46. Muscle fiber contraction in diabetic muscle
  47. Possible Role of Interaction between PPARαand Cyclophilin D in Cardioprotection of AMPK againstIn VivoIschemia-Reperfusion in Rats
  48. The Role of SIRT3 in Mediating Cardioprotective Effects of RAS Inhibition on Cardiac Ischemia-Reperfusion
  49. Mitochondria-targeted antioxidant preserves contractile properties and mitochondrial function of skeletal muscle in aged rats
  50. Cardiac Metabolic Pathways Affected in the Mouse Model of Barth Syndrome
  51. Regulation of Apoptotic Endonucleases by EndoG
  52. The calcium-ROS-pH triangle and mitochondrial permeability transition: challenges to mimic cardiac ischemia-reperfusion
  53. H9c2 and HL-1 cells demonstrate distinct features of energy metabolism, mitochondrial function and sensitivity to hypoxia-reoxygenation
  54. The beneficial effects of AMP kinase activation against oxidative stress are associated with prevention of PPARα-cyclophilin D interaction in cardiomyocytes
  55. Mitochondria-targeted ROS scavenger improves post-ischemic recovery of cardiac function and attenuates mitochondrial abnormalities in aged rats
  56. Inhibition of JNK Aggravates the Recovery of Rat Hearts after Global Ischemia: The Role of Mitochondrial JNK
  57. Crosstalk between mitogen-activated protein kinases and mitochondria in cardiac diseases: Therapeutic perspectives
  58. Downregulation of SIRT3 Exerts Mitochondrial Dysfunction in Cultured Cardiomyocytes
  59. Angiotensin II and Ischemic Preconditioning Synergize to Improve Mitochondrial Function While Showing Additive Effects on Ventricular Postischemic Recovery
  60. Letter to the editor: "Cyclosporin A in left ventricular remodeling after myocardial infarction"
  61. Crosstalk between AMPK activation and angiotensin II‐induced hypertrophy in cardiomyocytes: the role of mitochondria
  62. Mitochondria: the cell powerhouse and nexus of stress
  63. Cardiac-specific VLCAD deficiency induces dilated cardiomyopathy and cold intolerance
  64. Mitochondria: the cell powerhouse and nexus of stress
  65. Mitochondrial Permeability Transition and Cell Death: The Role of Cyclophilin D
  66. Cytoskeleton and regulation of mitochondrial function: the role of beta-tubulin II
  67. The Role of PPARα in Metformin-Induced Attenuation of Mitochondrial Dysfunction in Acute Cardiac Ischemia/Reperfusion in Rats
  68. Role of NF-κB and p38 MAPK activation in mediating angiotensin II and endothelin-1-induced stimulation in leptin production and cardiomyocyte hypertrophy
  69. Mitochondrial Permeability Transition: Known Phenomenon with Unknown Molecular Identity
  70. Direct Renin Inhibition Exerts an Anti-hypertrophic Effect Associated with Improved Mitochondrial Function in Post-infarction Heart Failure in Diabetic Rats
  71. Ouabain increases iNOS-dependent nitric oxide generation which contributes to the hypertrophic effect of the glycoside: possible role of peroxynitrite formation
  72. mTOR mediates RhoA-dependent leptin-induced cardiomyocyte hypertrophy
  73. Targeting the Mitochondrial Permeability Transition: Cardiac Ischemia-Reperfusion Versus Carcinogenesis
  74. Expression of mitochondrial fusion–fission proteins during post-infarction remodeling: the effect of NHE-1 inhibition
  75. Nitric oxide inhibits endothelin-1-induced neonatal cardiomyocyte hypertrophy via a RhoA-ROCK-dependent pathway
  76. Mitochondrial Permeability Transition Pore Opening as a Promising Therapeutic Target in Cardiac Diseases
  77. Anti-hypertrophic effect of NHE-1 inhibition involves GSK-3β-dependent attenuation of mitochondrial dysfunction
  78. Estrogen exerts concentration-dependent pro-and anti-hypertrophic effects on adult cultured ventricular myocytes. Role of NHE-1 in estrogen-induced hypertrophy
  79. The role of NHE-1 in myocardial hypertrophy and remodelling
  80. Leptin-induced cardiomyocyte hypertrophy involves selective caveolae and RhoA/ROCK-dependent p38 MAPK translocation to nuclei
  81. NHE-1 inhibition-induced cardioprotection against ischaemia/reperfusion is associated with attenuation of the mitochondrial permeability transition
  82. Actin Cytoskeleton Dynamics Promotes Leptin-Induced Vascular Smooth Muscle Hypertrophy via RhoA/ROCK- and Phosphatidylinositol 3-Kinase/Protein Kinase B-Dependent Pathways
  83. Mitochondrial Permeability Transition Pore Opening as an Endpoint to Initiate Cell Death and as a Putative Target for Cardioprotection
  84. Essential role of Rho/ROCK-dependent processes and actin dynamics in mediating leptin-induced hypertrophy in rat neonatal ventricular myocytes
  85. Distinct KATP Channels Mediate the Antihypertrophic Effects of Adenosine Receptor Activation in Neonatal Rat Ventricular Myocytes
  86. An autocrine role for leptin in mediating the cardiomyocyte hypertrophic effects of angiotensin II and endothelin-1
  87. NHE-1 inhibition improves cardiac mitochondrial function through regulation of mitochondrial biogenesis during postinfarction remodeling
  88. A60. Inhibition of NHE-1 attenuates MAPKs upregulation in hypertrophied car-diomyocytes through mitochondria
  89. A106. PPAR_α activation is associated with angiotensin II induced leptin production in cardiomyocyte mediated by JAK_STAT
  90. A30. Identification of KATP channels as mediators of antihypertrophic effects of adenosine receptor activation
  91. Antihypertrophic Effect of Na+/H+ Exchanger Isoform 1 Inhibition Is Mediated by Reduced Mitogen-Activated Protein Kinase Activation Secondary to Improved Mitochondrial Integrity and Decreased Generation of Mitochondrial-Derived Reactive Oxygen Species
  92. Leptin Induces Vascular Smooth Muscle Cell Hypertrophy through Angiotensin II- and Endothelin-1-Dependent Mechanisms and Mediates Stretch-Induced Hypertrophy
  93. NHE-1 inhibition improves impaired mitochondrial permeability transition and respiratory function during postinfarction remodelling in the rat
  94. Analysis of Erythrocyte and Platelet Membrane Proteins in Various Forms of  -Thalassemia
  95. Mitochondrial permeability transition pore opening during myocardial reperfusion—a target for cardioprotection
  96. Ischaemic Preconditioning Inhibits Opening of Mitochondrial Permeability Transition Pores in the Reperfused Rat Heart
  97. The effects of ischaemic preconditioning, diazoxide and 5‐hydroxydecanoate on rat heart mitochondrial volume and respiration
  98. Diazoxide and Ischaemic Preconditioning Act Differently on the Mitochondria: Is the Mitokatp Channel a Red Herring?
  99. Protection of hearts from reperfusion injury by propofol is associated with inhibition of the mitochondrial permeability transition
  100. Elucidating the molecular mechanism of the permeability transition pore and its role in reperfusion injury of the heart
  101. Effect of global ischemia on calcium sensitivity of diabetic rat hearts
  102. Cardiac performance and lipid metabolism of alloxan-induced diabetic rats: Effect of global ischemia
  103. Quantitative evaluation of relationship between cardiac energy metabolism and post-ischemic recovery of contractile function
  104. Protection of ischemic myocardium by exogenous phosphocreatine
  105. Biochemical aspects of the protective action of phosphocreatine on the ischemicmyocardium
  106. Contents of high energy phosphates and rates of mitochondrial oxidative reactions: Significance for cardiac contractile function
  107. Effect of phosphocreatine and related compounds on the phospholipid metabolism of ischemic heart
  108. Analysis of the antiarrhythmic action of phosphocreatine in acute myocardial ischemia in the dog
  109. The Permeability Transition Pore in Myocardial Ischemia and Reperfusion