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  1. BadR directly represses the expression of the glycerol utilization operon in the Lyme disease pathogen
  2. YebC Modulates OspC and VlsE Inverse Regulation and VlsE Expression in Persistent Lyme Disease
  3. The Rrp2-RpoN-RpoS pathway plays an important role in the blood-brain barrier transmigration of the Lyme disease pathogen
  4. Borrelia burgdorferic-di-AMP induces type I IFN response in macrophage through the activation of STING signaling pathway
  5. Host blood meal identity modifies vector gene expression and competency
  6. Gene Regulation and Transcriptomics
  7. Gene Regulation and Transcriptomics
  8. Role of HK2 in the Enzootic Cycle of Borrelia burgdorferi
  9. YebC regulates variable surface antigen VlsE expression and is required for host immune evasion in Borrelia burgdorferi
  10. Comparative vector competence of North American Lyme disease vectors
  11. Protective Immunity and New Vaccines for Lyme Disease
  12. Positive and Negative Regulation of Glycerol Utilization by the c-di-GMP Binding Protein PlzA in Borrelia burgdorferi
  13. The oligopeptide ABC transporter OppA4 negatively regulates the virulence factor OspC production of the Lyme disease pathogen
  14. The sigma factor σ54 is required for the long‐term survival of Leptospira biflexa in water
  15. Identification of Acetylated Proteins in Borrelia burgdorferi
  16. The EbpA-RpoN Regulatory Pathway of the Pathogen Leptospira interrogans Is Essential for Survival in the Environment
  17. Insight into the Dual Functions of Bacterial Enhancer-Binding Protein Rrp2 of Borrelia burgdorferi
  18. Outer Surface Protein OspC Is an Antiphagocytic Factor That Protects Borrelia burgdorferi from Phagocytosis by Macrophages
  19. Prioritizing drug targets in Clostridium botulinum with a computational systems biology approach
  20. DhhP, a Cyclic di-AMP Phosphodiesterase of Borrelia burgdorferi, Is Essential for Cell Growth and Virulence
  21. Cyclic Di-GMP Receptor PlzA Controls Virulence Gene Expression through RpoS in Borrelia burgdorferi
  22. Manganese and Zinc Regulate Virulence Determinants in Borrelia burgdorferi
  23. Rrp2, a Prokaryotic Enhancer-Like Binding Protein, Is Essential for Viability of Borrelia burgdorferi
  24. Role of Acetyl-Phosphate in Activation of the Rrp2-RpoN-RpoS Pathway in Borrelia burgdorferi
  25. Role of the Surface Lipoprotein BBA07 in the Enzootic Cycle of Borrelia burgdorferi
  26. Characterization of the Highly Regulated Antigen BBA05 in the Enzootic Cycle of Borrelia burgdorferi
  27. Rrp2, a σ54-Dependent Transcriptional Activator of Borrelia burgdorferi, Activates rpoS in an Enhancer-Independent Manner
  28. Abrogation of ospAB constitutively activates the Rrp2‐RpoN‐RpoS pathway (sigmaN‐sigmaS cascade) in Borrelia burgdorferi
  29. Essential Role of the Response Regulator Rrp2 in the Infectious Cycle of Borrelia burgdorferi
  30. Identification of Rrp2‐Controlled Mammalian Infection‐Associated Proteins in Borrelia Burgdorferi
  31. The Lyme disease agent exploits a tick protein to infect the mammalian host
  32. Analysis of the ospC Regulatory Element Controlled by the RpoN-RpoS Regulatory Pathway in Borrelia burgdorferi
  33. Essential Role for OspA/B in the Life Cycle of the Lyme Disease Spirochete
  34. The response regulator Rrp2 is essential for the expression of major membrane lipoproteins in Borrelia burgdorferi