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  1. B Cell-Specific Expression of Ataxia-Telangiectasia Mutated Protein Kinase Promotes Chronic Gammaherpesvirus Infection
  2. Interferon Regulatory Factor 1 and Type I Interferon Cooperate To Control Acute Gammaherpesvirus Infection
  3. Type I Interferon Counteracts Antiviral Effects of Statins in the Context of Gammaherpesvirus Infection
  4. Tumor Suppressor Interferon-Regulatory Factor 1 Counteracts the Germinal Center Reaction Driven by a Cancer-Associated Gammaherpesvirus
  5. Interferon Regulatory Factor 1 Restricts Gammaherpesvirus Replication in Primary Immune Cells
  6. Primary Macrophages Rely on Histone Deacetylase 1 and 2 Expression To Induce Type I Interferon in Response to Gammaherpesvirus Infection
  7. Mouse gammaherpesvirus-68 infection acts as a rheostat to set the level of type I interferon signaling in primary macrophages
  8. A Conserved Gammaherpesvirus Protein Kinase Targets Histone Deacetylases 1 and 2 To Facilitate Viral Replication in Primary Macrophages
  9. Modulation of B-cell tolerance by Murine Gammaherpesvirus 68 infection: requirement for Orf73 viral gene expression and follicular helper T cells
  10. Reply to "Testing for Herpesvirus Infection Is Essential in Children with Chromosomal-Instability Syndromes"
  11. Regulation of Antiviral CD8 T-Cell Responses
  12. Dynamic association of gammaherpesvirus DNA with core histone during de novo lytic infection of primary cells
  13. Gammaherpesvirus gene expression and DNA synthesis are facilitated by viral protein kinase and histone variant H2AX
  14. Conserved gammaherpesvirus kinase and histone variant H2AX facilitate gammaherpesvirus latency in vivo
  15. MHV68 complement regulatory protein facilitates MHV68 replication in primary macrophages in a complement independent manner
  16. Adenovirus E1A and E1B-19K proteins protect human hepatoma cells from transforming growth factor β1-induced apoptosis
  17. Adenovirus DNA binding protein inhibits SrCap-activated CBP and CREB-mediated transcription
  18. Radiation increases the activity of oncolytic adenovirus cancer gene therapy vectors that overexpress the ADP (E3-11.6K) protein