All Stories

  1. NCCR Chemical Biology: Interdisciplinary Research Excellence, Outreach, Education, and New Tools for Switzerland
  2. Retinoic acid induces expression of SLP-76: Expression with c-FMS enhances ERK activation and retinoic acid-induced differentiation/G0 arrest of HL-60 cells
  3. Retinoic acid-induced CD38 expression in HL-60 myeloblastic leukemia cells regulates cell differentiation or viability depending on expression levels
  4. RETINOIC ACID, BROMODEOXYURIDINE, AND THE Δ205 MUTANT POLYOMA VIRUS MIDDLE T ANTIGEN REGULATE EXPRESSION LEVELS OF A COMMON ENSEMBLE OF PROTEINS ASSOCIATED WITH EARLY STAGES OF INDUCING HL-60 LEUKEMIC CELL DIFFERENTIATION
  5. Retinoic acid causes MEK-dependent RAF phosphorylation through RARalpha plus RXR activation in HL-60 cells
  6. NONGENOMIC VITAMIN D3 ANALOGS ACTIVATING ERK2 IN HL-60 CELLS SHOW THAT RETINOIC ACID–INDUCED DIFFERENTIATION AND CELL CYCLE ARREST REQUIRE EARLY CONCURRENT MAPK AND RAR AND RXR ACTIVATION
  7. RETINOIC ACID INCREASES AMOUNT OF PHOSPHORYLATED RAF; ECTOPIC EXPRESSION OF cFMS REVEALS THAT RETINOIC ACID-INDUCED DIFFERENTIATION IS MORE STRONGLY DEPENDENT ON ERK2 SIGNALING THAN INDUCED G0 ARREST IS
  8. RETINOIC ACID INCREASES AMOUNT OF PHOSPHORYLATED RAF; ECTOPIC EXPRESSION OF cFMS REVEALS THAT RETINOIC ACID-INDUCED DIFFERENTIATION IS MORE STRONGLY DEPENDENT ON ERK2 SIGNALING THAN INDUCED G0 ARREST IS
  9. Retinoic acid selectively activates the ERK2 but not JNK/SAPK or P38 map kinases when inducing myeloid differentiation
  10. Transformation-Defective Polyoma Middle T Antigen Mutants Defective in PLCγ, PI-3, or src Kinase Activation Enhance ERK2 Activation and Promote Retinoic Acid-Induced, Cell Differentiation Like Wild-Type Middle T
  11. Polyoma Middle T Antigen in HL-60 Cells Accelerates Hematopoietic Myeloid and Monocytic Cell Differentiation
  12. FMS (CSF-1 Receptor) Prolongs Cell Cycle and Promotes Retinoic Acid-Induced Hypophosphorylation of Retinoblastoma Protein, G1 Arrest, and Cell Differentiation
  13. The Ratio of Retinoblastoma (RB) to fos and RB to myc Expression during the Cell Cycle
  14. RB phosphorylation in sodium butyrate-resistant HL-60 cells: Cross-resistance to retinoic acid but not vitamin D3
  15. DMSO, sodium butyrate, and TPA induce hypophosphorylation of RB with HL-60 cell differentiation
  16. Enhanced Cell Differentiation When RB Is Hypophosphorylated and Down-Regulated by Radicicol, a SRC-Kinase Inhibitor
  17. Late Dephosphorylation of the RB Protein in G2 during the Process of Induced Cell Differentiation
  18. Vitamin-Regulated Retinoblastoma Tumor Suppressor Gene Expression in Leukemic Cells
  19. C-FMS dependent HL-60 cell differentiation and regulation of RB gene expression
  20. 1,25-dihydroxy vitamin D3 and 12-O-tetradecanoyl phorbol-13-acetate synergistically induce monocytic cell differentiation: FOS and RB expression
  21. RB tumor suppressor gene expression responds to DNA synthesis inhibitors
  22. 42. Tumor suppressor gene expression responds to chemotherapeutic drugs
  23. Regulated expression of the RB “tumor suppressor gene” in normal lymphocyte mitogenesis: Elevated expression in transformed leukocytes and role as a “status quo” gene
  24. HTLV-III infection of EBV-genome-positive B-lymphoid cells with or without detectable T4 antigens
  25. Isolation of a new virus, HBLV, in patients with lymphoproliferative disorders