All Stories

  1. Substance P in heart failure: The good and the bad
  2. Substance P acting via the neurokinin-1 receptor regulates adverse myocardial remodeling in a rat model of hypertension
  3. Estrogen modulates the influence of cardiac inflammatory cells on function of cardiac fibroblasts
  4. Alpha-calcitonin gene-related peptide is protective against pressure overload-induced heart failure
  5. Could interferon-gamma be a therapeutic target for treating heart failure?
  6. Gender differences in non-ischemic myocardial remodeling: are they due to estrogen modulation of cardiac mast cells and/or membrane type 1 matrix metalloproteinase
  7. Stem cell factor is responsible for the rapid response in mature mast cell density in the acutely stressed heart
  8. The Convergence of Ancient Chinese Medicine With Modern Therapeutics to Prevent Cardiac Fibrosis
  9. Prevention of adverse cardiac remodeling to volume overload in female rats is the result of an estrogen-altered mast cell phenotype
  10. Substance P induces adverse myocardial remodelling via a mechanism involving cardiac mast cells
  11. Tryptase/Protease-Activated Receptor 2 Interactions Induce Selective Mitogen-Activated Protein Kinase Signaling and Collagen Synthesis by Cardiac Fibroblasts
  12. Cardiovascular Changes During Maturation and Ageing in Male and Female Spontaneously Hypertensive Rats
  13. Cardiac mast cells: the centrepiece in adverse myocardial remodelling
  14. Oxidative stress-mediated cardiac mast cell degranulation
  15. Interleukin 6 Mediates Myocardial Fibrosis, Concentric Hypertrophy, and Diastolic Dysfunction in Rats
  16. Sympathetic Nervous System Modulation of Inflammation and Remodeling in the Hypertensive Heart
  17. TNF-  inhibition attenuates adverse myocardial remodeling in a rat model of volume overload
  18. Cardiac Mast Cells Mediate Left Ventricular Fibrosis in the Hypertensive Rat Heart
  19. Regulation of matrix metalloproteinases is at the heart of myocardial remodeling
  20. Protection from adverse myocardial remodeling secondary to chronic volume overload in mast cell deficient rats
  21. A novel technique for isolating functional mast cells from the heart
  22. Response of cardiac mast cells to atrial natriuretic peptide
  23. Inhibition of matrix metalloproteinase activity by ACE inhibitors prevents left ventricular remodeling in a rat model of heart failure
  24. Arachidonic Acid Metabolism as a Potential Mediator of Cardiac Fibrosis Associated with Inflammation
  25. Antifibrotic Activity of an Inhibitor of Group IIA Secretory Phospholipase A2 in Young Spontaneously Hypertensive Rats
  26. Reversal of cardiac dysfunction by selective ET‐A receptor antagonism
  27. Increased Calcium Influx Mediates Increased Cardiac Stiffness in Hyperthyroid Rats