All Stories

  1. Dextromethorphan Is a Novel Pharmacological Inhibitor of F 1 F O ‐ ...
  2. Mitochondrial Permeability Transition Pore: The Cardiovascular Disease’s Molecular Achilles Heel
  3. Reprogramming the Mitochondrion in Atherosclerosis: Targets for Vascular Protection
  4. Inflammatory mitochondrial signalling and viral mimicry in cancer
  5. Oxygen Deprivation and Brain Inflammation: The Mitochondrial Link to Alzheimer's Progression
  6. Mitochondrial bioenergetics analysis on SLC-selected boar spermatozoa during liquid storage
  7. Analysis of stallion spermatozoa metabolism using Agilent Seahorse XFp Technology
  8. Mitochondrial sirtuin 3 and role of natural compounds: the effect of post-translational modifications on cellular metabolism
  9. UCP2, a Member of the Mitochondrial Uncoupling Proteins: An Overview from Physiological to Pathological Roles
  10. H+‐slip correlated to rotor free‐wheeling as cause of F1FO‐ATPase dysfunction in primary mitochondrial disorders
  11. Selenite ameliorates the ATP hydrolysis of mitochondrial F1FO-ATPase by changing the redox state of thiol groups and impairs the ADP phosphorylation
  12. Understanding differential aspects of microdiffusion (channeling) in the Coenzyme Q and Cytochrome c regions of the mitochondrial respiratory system
  13. Mineralocorticoid Receptor Antagonism Prevents Type 2 Familial Partial Lipodystrophy Brown Adipocyte Dysfunction
  14. Proton leak through the UCPs and ANT carriers and beyond: A breath for the electron transport chain
  15. Inflammatory signaling in NASH driven by hepatocyte mitochondrial dysfunctions
  16. Cell bioenergetics and ATP production of boar spermatozoa
  17. Cell Metabolism Therapy by Small Natural Compounds
  18. Two separate pathways underlie NADH and succinate oxidation in swine heart mitochondria: Kinetic evidence on the mobile electron carriers
  19. Inflammation, Mitochondria and Natural Compounds Together in the Circle of Trust
  20. Novel Regioselective Synthesis of 1,3,4,5-Tetrasubstituted Pyrazoles and Biochemical Valuation on F1FO-ATPase and Mitochondrial Permeability Transition Pore Formation
  21. ‘Rotor free-wheeling’ in impaired F1FO-ATPase induces congenital hypermetabolism
  22. GPR35, ally of the anti-ischemic ATPIF1-ATP synthase interaction
  23. Effects of cryopreservation on the mitochondrial bioenergetics of bovine sperm
  24. The Impairment of Cell Metabolism by Cardiovascular Toxicity of Doxorubicin Is Reversed by Bergamot Polyphenolic Fraction Treatment in Endothelial Cells
  25. Cellular Metabolism and Bioenergetic Function in Human Fibroblasts and Preadipocytes of Type 2 Familial Partial Lipodystrophy
  26. What happens when the mitochondrial H+-translocating F1FO-ATP(hydrol)ase becomes a molecular target of calcium? The pore opens
  27. Protein folding and unfolding: proline cis‐trans isomerization at the c subunits of F1FO‐ATPase might open a high conductance ion channel
  28. Mitochondria Bioenergetic Functions and Cell Metabolism Are Modulated by the Bergamot Polyphenolic Fraction
  29. From the Structural and (Dys)Function of ATP Synthase to Deficiency in Age-Related Diseases
  30. From the Structural and (Dys)function of ATP Synthase to Deficiency in Age-Related Diseases
  31. Impaired Mitochondrial Bioenergetics under Pathological Conditions
  32. Protein folding and unfolding: proline cis - trans isomerization at the c subunits of F 1 F O -ATPase might open a high conductance ion channel
  33. Unraveling Energy Substrate Utilization in Sperm Cells
  34. Enjoy your journey: the bergamot polyphenols from the tree to the cell metabolism
  35. The mitochondrial F1FO-ATPase exploits the dithiol redox state to modulate the permeability transition pore
  36. SARS‐CoV‐2 first contact: Spike–ACE2 interactions in COVID‐19
  37. Sulfide affects the mitochondrial respiration, the Ca2+-activated F1FO-ATPase activity and the permeability transition pore but does not change the Mg2+-activated F1FO-ATPase activity in swine heart mitochondria
  38. Relationship between serum concentration, functional parameters and cell bioenergetics in IPEC-J2 cell line
  39. Molecular and Supramolecular Structure of the Mitochondrial Oxidative Phosphorylation System: Implications for Pathology
  40. Biological characteristics and metabolic profile of canine mesenchymal stem cells isolated from adipose tissue and umbilical cord matrix
  41. Ca2+ as cofactor of the mitochondrial H+‐translocating F1FO‐ATP(hydrol)ase
  42. Mitochondrial F1FO-ATPase and permeability transition pore response to sulfide in the midgut gland of Mytilus galloprovincialis
  43. Incoming news on the F-type ATPase structure and functions in mammalian mitochondria
  44. 1,5‐Disubstituted‐1,2,3‐triazoles as inhibitors of the mitochondrial Ca 2+ ‐activated F 1 F O ‐ATP(hydrol)ase and the permeability transition pore
  45. Effects of Hydrogen Sulfide Donor NaHS on Porcine Vascular Wall-Mesenchymal Stem Cells
  46. Mitochondrial F-type ATP synthase: multiple enzyme functions revealed by the membrane-embedded FO structure
  47. Nicotinamide Nucleotide Transhydrogenase as a Sensor of Mitochondrial Biology
  48. The mitochondrial permeability transition pore in cell death: A promising drug binding bioarchitecture
  49. Mitochondrial Ca 2+ ‐activated F 1 F O ‐ATPase hydrolyzes ATP and promotes the permeability transition pore
  50. Emerging Roles for the Mitochondrial ATP Synthase Supercomplexes
  51. A Therapeutic Role for the F1FO-ATP Synthase
  52. Characterization of metabolic profiles and lipopolysaccharide effects on porcine vascular wall mesenchymal stem cells
  53. Crucial aminoacids in the FO sector of the F1FO-ATP synthase address H+ across the inner mitochondrial membrane: molecular implications in mitochondrial dysfunctions
  54. Lipid-protein interactions in mitochondrial membranes from bivalve mollusks: molecular strategies in different species
  55. From the Ca 2+ -activated F 1 F O -ATPase to the mitochondrial permeability transition pore: an overview
  56. A Lethal Channel between the ATP Synthase Monomers
  57. New insight in a new entity: the mitochondrial permeability transition pore arises from the Ca 2+ -activated F 1 F O -ATPases
  58. The inhibition of the mitochondrial F1FO-ATPase activity when activated by Ca2+ opens new regulatory roles for NAD+
  59. Mitochondrial permeability transition, F 1 F O ‐ATPase and calcium: an enigmatic triangle
  60. Post-translational modifications of the mitochondrial F 1 F O -ATPase
  61. A preliminary study on a novel sea water disinfection process by a peroxy-acid compound to complement and improve the microbial depuration of clams ( Ruditapes philippinarum )
  62. Kinetic properties of the mitochondrial F 1 F O -ATPase activity elicited by Ca 2+ in replacement of Mg 2+
  63. Mitochondrial permeability transition, F 1 F O ‐ATPase and calcium: an enigmatic triangle
  64. Glucose and glutamine in the mitochondrial oxidative metabolism of stem cells
  65. Mercury and protein thiols: Stimulation of mitochondrial F1FO-ATPase and inhibition of respiration
  66. Preferential nitrite inhibition of the mitochondrial F1FO-ATPase activities when activated by Ca2+ in replacement of the natural cofactor Mg2+
  67. Lipid unsaturation per se does not explain the physical state of mitochondrial membranes in Mytilus galloprovincialis
  68. Thiol-Related Regulation of the Mitochondrial F1FO-ATPase Activity
  69. The c-Ring of the F1FO-ATP Synthase: Facts and Perspectives
  70. Opposite Rotation Directions in the Synthesis and Hydrolysis of ATP by the ATP Synthase: Hints from a Subunit Asymmetry
  71. Long-chain PUFA enrichment in microalgae and metabolic dynamics inTapes philippinarumlarvae
  72. The a subunit asymmetry dictates the two opposite rotation directions in the synthesis and hydrolysis of ATP by the mitochondrial ATP synthase
  73. Thiol oxidation of mitochondrial FO-c subunits: A way to switch off antimicrobial drug targets of the mitochondrial ATP synthase
  74. Thiol oxidation is crucial in the desensitization of the mitochondrial F1FO-ATPase to oligomycin and other macrolide antibiotics
  75. The mitochondrial F1FO-ATPase desensitization to oligomycin by tributyltin is due to thiol oxidation
  76. Modifiers of the oligomycin sensitivity of the mitochondrial F1F0-ATPase
  77. Mussel and mammalian ATP synthase share the same bioenergetic cost of ATP
  78. Toxicity of organotin compounds: Shared and unshared biochemical targets and mechanisms in animal cells
  79. Modulation of the F1FO-ATPase function by butyltin compounds
  80. Dietary Enhancement of Selected Fatty Acid Biosynthesis in the Digestive Gland of Mytilus galloprovincialis Lmk.
  81. Tri-n-butyltin binding to a low-affinity site decreases the F1FO-ATPase sensitivity to oligomycin in mussel mitochondria
  82. Organotin Effects in Different Phyla: Discrepancies and Similarities
  83. Tributyltin-driven enhancement of the DCCD insensitive Mg-ATPase activity in mussel digestive gland mitochondria
  84. Structural and functional changes in gill mitochondrial membranes from the Mediterranean mussel Mytilus galloprovincialis exposed to tri-n-butyltin
  85. Multi-site TBT binding skews the inhibition of oligomycin on the mitochondrial Mg–ATPase in Mytilus galloprovincialis
  86. Tributyltin (TBT) and mitochondrial respiration in mussel digestive gland
  87. Tributyltin (TBT) and dibutyltin (DBT) differently inhibit the mitochondrial Mg-ATPase activity in mussel digestive gland
  88. Tributyltin inhibits the oligomycin-sensitive Mg-ATPase activity in Mytilus galloprovincialis digestive gland mitochondria
  89. Tributyltin (TBT) inhibition of oligomycin-sensitive Mg-ATPase activity in mussel mitochondria