All Stories

  1. hnRNP L is essential for myogenic differentiation and modulates myotonic dystrophy pathologies
  2. Correction: RSK phosphorylates SOS1 creating 14-3-3-docking sites and negatively regulating MAPK activation
  3. Megf10 deficiency impairs skeletal muscle stem cell migration and muscle regeneration
  4. AAV9-TAZ Gene Replacement Ameliorates Cardiac TMT Proteomic Profiles in a Mouse Model of Barth Syndrome
  5. Selective serotonin reuptake inhibitors ameliorate MEGF10 myopathy
  6. The impact of Megf10/Drpr gain‐of‐function on muscle development in Drosophila
  7. AAV-MediatedTAZGene Replacement Restores Mitochondrial and Cardioskeletal Function in Barth Syndrome
  8. Impact of PYROXD1 deficiency on cellular respiration and correlations with genetic analyses of limb-girdle muscular dystrophy in Saudi Arabia and Sudan
  9. Consequences of MEGF10 deficiency on myoblast function and Notch1 interactions
  10. Mitogen-activated protein kinase phosphatase-3 (MKP-3) in the surgical wound is necessary for the resolution of postoperative pain in mice
  11. RSK-dependent 14-3-3 interactions
  12. Mitogen-activated protein kinase (MAPK) phosphatase-3 (MKP-3) displays a p-JNK-MAPK substrate preference in astrocytes in vitro
  13. (283) Spinal cord MKP-3/MAPK signaling impairment results in the transition from acute to chronic postoperative pain
  14. Spinal Mitogen-Activated Protein Kinase Phosphatase-3 (MKP-3) is Necessary for the Normal Resolution of Mechanical Allodynia in a Mouse Model of Acute Postoperative Pain
  15. Proteomic analysis of the cilia membrane of Paramecium tetraurelia
  16. RSK phosphorylates SOS1 creating 14-3-3-docking sites and negatively regulating MAPK activation