All Stories

  1. The fundamental role of mitochondria–endoplasmic reticulum contacts in ageing and declining healthspan
  2. Emerging and Novel Therapeutic Treatments Targeting Mitochondrial-Endoplasmic Reticulum Contact Sites in Metabolic and Vascular Disorders
  3. FLT4 causes developmental disorders of the cardiovascular and lymphovascular systems via pleiotropic molecular mechanisms
  4. Metabolism and proteostasis: at the intersection of inflammation- associated decline in healthspan
  5. BS29 FLT4 pleiotropy in congenital cardiac or lymphovascular disease is mediated through distinct cellular mechanisms
  6. The physiological and pathological functions of VEGFR3 in cardiac and lymphatic development and related diseases
  7. Scientific Business Abstracts of the 113th Annual Meeting of the Association of Physicians of Great Britain and Ireland
  8. BS9 KMT2C- a tetralogy of fallot candidate gene
  9. C Identification of the major genetic contributors to tetralogy of fallot
  10. 139 Identification of the major genetic contributors to tetralogy of fallot
  11. Whole Exome Sequencing Reveals the Major Genetic Contributors to Non-Syndromic Tetralogy of Fallot
  12. Deleterious genetic variants inNOTCH1are a major contributor to the incidence of non-syndromic Tetralogy of Fallot
  13. Mitochondrial Proteins Moonlighting in the Nucleus
  14. A nuclear sensor of mitochondrial function
  15. A nuclear role for the respiratory enzyme CLK-1 in regulating mitochondrial stress responses and longevity
  16. A new role for an old enemy