All Stories

  1. Elementary processes and mechanisms of nanopore formation induced by antimicrobial peptides and other membrane-active peptides
  2. Effect of membrane tension on (−)-epigallocatechin gallate-induced burst of single giant unilamellar vesicles
  3. Effect of membrane tension on pore formation induced by antimicrobial peptides and other membrane-active peptides
  4. Single-cell analysis of antimicrobial compound–induced cell death of bacterial cells
  5. Effect of osmotic pressure on membrane permeation through antimicrobial peptide-induced pores
  6. Interaction of peptides/proteins/bioactive compounds with single giant unilamellar vesicles under more biological conditions
  7. Relationship between oligoarginine-induced membrane damage of single Escherichia coli cells and entry of the peptide into the cytoplasm
  8. Processes and mechanisms underlying burst of giant unilamellar vesicles induced by antimicrobial peptides and compounds
  9. Effect of Phosphatidylethanolamine on Pore Formation Induced by the Antimicrobial Peptide PGLa
  10. Effect of membrane tension on antimicrobial peptide PGLa-induced pore formation in lipid bilayers
  11. Estimation of negative membrane tension in lipid bilayers and its effect on antimicrobial peptide magainin 2-induced pore formation
  12. Relationship between antimicrobial peptides-induced cell membrane damage and bactericidal activity
  13. Antimicrobial peptide magainin 2-induced rupture of single giant unilamellar vesicles comprising E. coli polar lipids
  14. Effect of monolayer spontaneous curvature on constant tension-induced pore formation in lipid bilayers
  15. Role of interfacial hydrophobicity in antimicrobial peptide magainin 2-induced nanopore formation
  16. Single-Cell Analysis of the Antimicrobial and Bactericidal Activities of the Antimicrobial Peptide Magainin 2
  17. Effect of osmotic pressure on pore formation in lipid bilayers by the antimicrobial peptide magainin 2
  18. Sulfur-doped carbon dots@polydopamine-functionalized magnetic silver nanocubes for dual-modality detection of norovirus
  19. A Single GUV Method for Revealing the Action of Cell-Penetrating Peptides in Biomembranes
  20. Translocation and entry of label-free AMP, PGLa, without pore formation
  21. Membrane potential enhances the entry of a short AMP into bacterial cells and lipid vesicles
  22. Fluorescent and electrochemical dual-mode detection of Chikungunya virus E1 protein using fluorophore-embedded and redox probe-encapsulated liposomes
  23. Effect of membrane potential on pore formation by the antimicrobial peptide magainin 2 in lipid bilayers
  24. Estimation of membrane tension in a GUV in a buffer containing a physiological conc. of ions
  25. Use of Target-Specific Liposome and Magnetic Nanoparticle Conjugation for the Amplified Detection of Norovirus
  26. Development of a new method to detect the entry of label-free peptides into vesicle lumen
  27. Effect of membrane potential on activities of AMPs and CPPs revealed by the single GUV method
  28. Membrane potential enhances the rate of entry of cell-penetrating peptides (CPPs) into the GUV lumen
  29. Membrane potential enhances membrane damage induced by antimicrobial peptide lactoferricin B
  30. The role of membrane tension in the action of antimicrobial peptides and cell-penetrating peptides in biomembranes
  31. Asymmetric distribution of lipids and peptides induces asymmetric lipid packing and membrane tension
  32. Elementary Processes and Mechanisms of Interactions of Antimicrobial Peptides with Membranes—Single Giant Unilamellar Vesicle Studies—
  33. Elementary processes of antimicrobial peptide PGLa-induced pore formation in lipid bilayers
  34. Membrane tension enhances the rate of transbilayer diffusion of lipid molecules
  35. Continuous detection of entry of cell-penetrating peptide transportan 10 into single vesicles
  36. Elementary processes for the entry of cell-penetrating peptides into lipid bilayer vesicles and bacterial cells
  37. Mechanism of Initial Stage of Pore Formation Induced by Antimicrobial Peptide Magainin 2
  38. Low-pH-Induced Lamellar to Bicontinuous Primitive Cubic Phase Transition in Dioleoylphosphatidylserine/Monoolein Membranes
  39. Entry of a Six-Residue Antimicrobial Peptide Derived from Lactoferricin B into Single Vesicles and Escherichia coli Cells without Damaging their Membranes
  40. Experimental Estimation of Membrane Tension Induced by Osmotic Pressure
  41. Effects of Mechanical Properties of Lipid Bilayers on the Entry of Cell-Penetrating Peptides into Single Vesicles
  42. Effects of Lipid Compositions on the Entry of Cell Penetrating Peptide Oligoarginine into Single Vesicles
  43. Experimental Estimation of Membrane Tension Induced by Osmotic Pressure
  44. Effects of Lipid Composition on the Entry of Cell-Penetrating Peptide Oligoarginine into Single Vesicles
  45. Activation Energy of the Low-pH-Induced Lamellar to Bicontinuous Cubic Phase Transition in Dioleoylphosphatidylserine/Monoolein
  46. Analysis of constant tension-induced rupture of lipid membranes using activation energy
  47. Antimicrobial Peptide Lactoferricin B-Induced Rapid Leakage of Internal Contents from Single Giant Unilamellar Vesicles
  48. Communication: Activation energy of tension-induced pore formation in lipid membranes
  49. Electrostatic interaction effects on tension-induced pore formation in lipid membranes
  50. Stretch-Activated Pore of the Antimicrobial Peptide, Magainin 2
  51. Initial Step of pH-Jump-Induced Lamellar to Bicontinuous Cubic Phase Transition in Dioleoylphosphatidylserine/Monoolein
  52. A Model for Targeting Colon Carcinoma Cells Using Single-Chain Variable Fragments Anchored on Virus-Like Particles via Glycosyl Phosphatidylinositol Anchor
  53. Entry of Cell-Penetrating Peptide Transportan 10 into a Single Vesicle by Translocating Across Lipid Membrane and Its Induced Pores
  54. The single GUV method for revealing the functions of antimicrobial, pore-forming toxin, and cell-penetrating peptides or proteins
  55. Rate Constant of Tension-Induced Pore Formation in Lipid Membranes
  56. The Single-Giant Unilamellar Vesicle Method Reveals Lysenin-Induced Pore Formation in Lipid Membranes Containing Sphingomyelin
  57. A membrane filtering method for the purification of giant unilamellar vesicles
  58. Kinetics of low pH-induced lamellar to bicontinuous cubic phase transition in dioleoylphosphatidylserine/monoolein
  59. Spontaneous insertion of lipopolysaccharide into lipid membranes from aqueous solution
  60. Kinetic Pathway of Antimicrobial Peptide Magainin 2-Induced Pore Formation in Lipid Membranes
  61. 単一GUV法による抗菌ペプチドのポア形成の研究
  62. The size of the pore in lipid membranes induced by antimicrobial peptide magainin 2
  63. Magainin 2-Induced Pore Formation in the Lipid Membranes Depends on Its Concentration in the Membrane Interface
  64. Chapter 7 Transformation Between Liposomes and Cubic Phases of Biological Lipid Membranes Induced by Modulation of Electrostatic Interactions
  65. Water permeability of lipid membranes of GUVs and its dependence on actin cytoskeletons inside the GUVs
  66. High affinity Zn2+ inhibitory site(s) for the trypsin-like peptidase of the 20S proteasome
  67. Low-pH-Induced Transformation of Bilayer Membrane into Bicontinuous Cubic Phase in Dioleoylphosphatidylserine/Monoolein Membranes
  68. Chapter 5 The Single Guv Method to Reveal Elementary Processes of Leakage of Internal Contents from Liposomes Induced by Antimicrobial Substances
  69. Effects of Surface Charge Density of Lipid Membranes on the Pore Formation Induced by Magainin 2
  70. Single GUV Method Reveals Interaction of Tea Catechin (−)-Epigallocatechin Gallate with Lipid Membranes
  71. Vesicle Fission of Giant Unilamellar Vesicles of Liquid-Ordered-Phase Membranes Induced by Amphiphiles with a Single Long Hydrocarbon Chain
  72. The “Le Chatelier's Principle”-Governed Response of Actin Filaments to Osmotic Stress
  73. Single Giant Unilamellar Vesicle Method Reveals Effect of Antimicrobial Peptide, Magainin 2, and Antibacterial Substance, Tea Catechin, on Membrane Permeability and Membrane Structure
  74. Single Giant Unilamellar Vesicle Method Reveals Effect of Antimicrobial Peptide Magainin 2 on Membrane Permeability
  75. Formation of Cubic Phases from Large Unilamellar Vesicles of Dioleoylphosphatidylglycerol/Monoolein Membranes Induced by Low Concentrations of Ca2+
  76. The effect of peptides and ions interacting with an electrically neutral membrane interface on the structure and stability of lipid membranes in the liquid-crystalline phase and in the liquid-ordered phase
  77. Cationic DMPC/DMTAP Lipid Bilayers:  Local Lateral Polarization of Phosphatidylcholine Headgroups
  78. Effect of Positively Charged Short Peptides on Stability of Cubic Phases of Monoolein/Dioleoylphosphatidic Acid Mixtures
  79. Design and Facile Synthesis of Neoglycolipids as Lactosylceramide Mimetics and Their Transformation into Glycoliposomes
  80. The Single GUV Method for Probing Biomembrane Structure and Function
  81. Stability of giant unilamellar vesicles and large unilamellar vesicles of liquid-ordered phase membranes in the presence of Triton X-100
  82. Shape Changes and Vesicle Fission of Giant Unilamellar Vesicles of Liquid-Ordered Phase Membrane Induced by Lysophosphatidylcholine
  83. Lipid Membrane Formation by Vesicle Fusion on Silicon Dioxide Surfaces Modified with Alkyl Self-Assembled Monolayer Islands
  84. Optical nanospectroscopy applications in material science
  85. Membrane Fusion of Giant Unilamellar Vesicles of Neutral Phospholipid Membranes Induced by La3+
  86. Low concentration of dioleoylphosphatidic acid induces an inverted hexagonal (HII) phase transition in dipalmitoleoylphosphatidylethanolamine membranes
  87. Low pH Stabilizes the Inverted Hexagonal II Phase in Dipalmitoleoylphosphatidylethanolamine Membrane
  88. Atomic force microscopy studies of interaction of the 20S proteasome with supported lipid bilayers
  89. Effect of de Novo Designed Peptides Interacting with the Lipid-Membrane Interface on the Stability of the Cubic Phases of the Monoolein Membrane
  90. Mechanical response of single filamin A (ABP-280) molecules and its role in the actin cytoskeleton
  91. Shape Changes of Giant Unilamellar Vesicles of Phosphatidylcholine Induced by a De Novo Designed Peptide Interacting with Their Membrane Interface
  92. La3+ and Gd3+ induce shape change of giant unilamellar vesicles of phosphatidylcholine
  93. A model of pressure-induced interdigitation of phospholipid membranes
  94. A new method for the preparation of giant liposomes in high salt concentrations and growth of protein microcrystals in them
  95. La3+ stabilizes the hexagonal II (HII) phase in phosphatidylethanolamine membranes
  96. Effect of Electrostatic Interactions on Phase Stability of Cubic Phases of Membranes of Monoolein/Dioleoylphosphatidic Acid Mixtures
  97. The mechanism of the stabilization of the hexagonal II (H II ) phase in phosphatidylethanolamine membranes in the presence of low concentrations of dimethyl sulfoxide
  98. Mechanical unfolding of single filamin A (ABP‐280) molecules detected by atomic force microscopy
  99. Low concentration of DMSO stabilizes the bilayer gel phase rather than the interdigitated gel phase in dihexadecylphosphatidylcholine membrane
  100. Effects of electrostatic interaction on the phase stability and structures of cubic phases of monoolein/oleic acid mixture membranes
  101. Effects of solvents interacting favorably with hydrophilic segments of the membrane surface of phosphatidylcholine on their gel-phase membranes in water
  102. Low pH Induces an Interdigitated Gel to Bilayer Gel Phase Transition in Dihexadecylphosphatidylcholine Membrane
  103. Intermembrane distance in multilamellar vesicles of phosphatidylcholine depends on the interaction free energy between solvents and the hydrophilic segments of the membrane surface
  104. Ion Permeability of a Membrane with Soft Polar Interfaces. 2. The Polar Zones as the Rate-Determining Step
  105. Phase transition between hexagonal II(HII) and liquid-crystalline phase induced by interaction between solvents and segments of the membrane surface of dioleoylphosphatidylethanolamine
  106. Osmotic stress induces a phase transition from interdigitated gel phase to bilayer gel phase in multilamellar vesicles of dihexadecylphosphatidylcholine
  107. Interaction of the surface of biomembrane with solvents: structure of multilamellar vesicles of dipalmitoylphosphatidylcholine in acetone-water mixtures
  108. Organic solvents induce interdigitated gel structures in multilamellar vesicles of dipalmitoylphosphatidylcholine
  109. Polymorphism of F-Actin Assembly. 1. A Quantitative Phase Diagram of F-Actin
  110. Formation of ion channels in lipid bilayers by a peptide with the predicted transmembrane sequence of botulinum neurotoxin A
  111. Direct evidence of induction of interdigitated gel structure in large unilamellar vesicles of dipalmitoylphosphatidylcholine by ethanol: studies by excimer method and high-resolution electron cryomicroscopy
  112. Effect of oligomers of ethylene glycol on thermotropic phase transition of dipalmitoylphosphatidylcholine multilamellar vesicles
  113. Phase transitions of phospholipid vesicles under osmotic stress and in the presence of ethylene glycol
  114. Studies of alcohol-induced interdigitated gel phase in phosphatidylcholine multilamellar vesicles by the excimer method
  115. Phase separation of triton X-100 micelle solution induced by osmotic stress
  116. Deformation and instability of membrane structure of phospholipid vesicles caused by osmophobic association: mechanical stress model for the mechanism of poly(ethylene glycol)-induced membrane fusion
  117. Poly(ethylene glycol)-induced shrinkage of Sephadex gel. A model system for quantitative analysis of osmoelastic coupling
  118. Osmoelastic coupling in biological structures: formation of parallel bundles of actin filaments in a crystalline-like structure caused by osmotic stress
  119. Osmoelastic coupling in biological structures: a comprehensive thermodynamic analysis of the osmotic response of phospholipid vesicles and a reevaluation of the "dehydration force" theory
  120. Osmoelastic coupling in biological structures: decrease in membrane fluidity and osmophobic association of phospholipid vesicles in response to osmotic stress
  121. Phase Transition in Di-oleoylphosphatidylglycerol/Monoolein Membranes due to Interactions of Positively Charged Peptides at their Lipid Membrane-Interface