All Stories

  1. Formation of a complex between TMEM217 and the sodium-proton exchanger SLC9C1 is crucial for mouse sperm motility and male fertility

    Article • Proceedings of the National Academy of Sciences, October 2025, Proceedings of the National Academy of Sciences

    masahito ikawa

  2. TEX38 localizes ZDHHC19 to the plasma membrane and regulates sperm head morphogenesis in mice

    Article • Proceedings of the National Academy of Sciences, March 2025, Proceedings of the National Academy of Sciences

    masahito ikawa

  3. 1700029I15Rik orchestrates the biosynthesis of acrosomal membrane proteins required for sperm–egg interaction

    Article • Proceedings of the National Academy of Sciences, February 2023, Proceedings of the National Academy of Sciences

    masahito ikawa

  4. Trim41 is required to regulate chromosome axis protein dynamics and meiosis in male mice

    Article • PLoS Genetics, June 2022, PLOS

    masahito ikawa, Professor Kei-ichiro Ishiguro

  5. SPATA33 localizes calcineurin to the mitochondria and regulates sperm motility in mice

    Article • Proceedings of the National Academy of Sciences, August 2021, Proceedings of the National Academy of Sciences

    masahito ikawa

  6. KCTD19 and its associated protein ZFP541 are independently essential for meiosis in male mice

    Article • PLoS Genetics, May 2021, PLOS

    masahito ikawa, Professor Kei-ichiro Ishiguro

  7. ARMC12 regulates spatiotemporal mitochondrial dynamics during spermiogenesis and is required for male fertility

    Article • Proceedings of the National Academy of Sciences, February 2021, Proceedings of the National Academy of Sciences

    masahito ikawa

  8. Nexin-Dynein regulatory complex component DRC7 but not FBXL13 is required for sperm flagellum formation and male fertility in mice

    Article • PLoS Genetics, January 2020, PLOS

    masahito ikawa

  9. New insights into the role of Jmjd3 and Utx in axial skeletal formation in mice

    Article • The FASEB Journal, February 2017, Federation of American Societies For Experimental Biology (FASEB)

    masahito ikawa

  10. CRISPR/Cas9-mediated mutation revealed cytoplasmic tail is dispensable for IZUMO1 function and male fertility

    Article • Reproduction, September 2016, Bioscientifica

    masahito ikawa

  11. Kidney-specific knockout ofSav1in the mouse promotes hyperproliferation of renal tubular epithelium through suppression of the Hippo pathway

    Article • The Journal of Pathology, March 2016, Wiley

    Dr Keiko Matsuura, masahito ikawa