All Stories

  1. Two Distinct Mechanisms Govern RpoS-Mediated Repression of Tick-Phase Genes during Mammalian Host Adaptation byBorrelia burgdorferi, the Lyme Disease Spirochete
  2. Borrelia burgdorferi induces a type I interferon response during early stages of disseminated infection in mice
  3. Borrelia burgdorferi Pathogenesis and the Immune Response
  4. Borrelia burgdorferi clinical isolates induce human innate immune responses that are not dependent on genotype
  5. Induction of indoleamine 2,3-dioxygenase by Borrelia burgdorferi in human immune cells correlates with pathogenic potential
  6. Induction of Type I and Type III Interferons by Borrelia burgdorferi Correlates with Pathogenesis and Requires Linear Plasmid 36
  7. Pathogenesis of Post-Lyme Disease Symptoms
  8. Borrelia burgdorferi RNA Induces Type I and III Interferons via Toll-Like Receptor 7 and Contributes to Production of NF- B-Dependent Cytokines
  9. Identification of OppA2 Linear Epitopes as Serodiagnostic Markers for Lyme Disease
  10. Borrelia burgdorferi Requires Glycerol for Maximum Fitness During The Tick Phase of the Enzootic Cycle
  11. Endosomal signaling by Lyme disease bacterium
  12. Borrelia burgdorferi bba74 Is Expressed Exclusively during Tick Feeding and Is Regulated by Both Arthropod- and Mammalian Host-Specific Signals
  13. Pattern of Proinflammatory Cytokine Induction in RAW264.7 Mouse Macrophages Is Identical for Virulent and Attenuated Borrelia burgdorferi
  14. Role of Immunoglobulin A in Protection against Reovirus Entry into Murine Peyer's Patches
  15. Schistosomamansoni gene GP22 encodes the tegumental antigen Sm25: (1) antibodies to a predicted B-cell epitope of Sm25 cross-react with other candidate vaccine worm antigens; (2) characterization of a recombinant product containing tandem-repeats of th...
  16. Evaluation of recombinant protein r140, a polypeptide segment of tegumental glycoprotein Sm25, as a defined antigen vaccine against Schistosoma mansoni
  17. Immobilization of Schistosoma mansoni miracidia by activation of the alternate complement pathway at unusually high serum dilution