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  1. The TGF-β1 Signaling Pathway as an Attractive Target in the Fibrosis Pathogenesis of Sjögren’s Syndrome
  2. Downstream activation of NF-κB in the EDA-A1/EDAR signalling in Sjögren's syndrome and its regulation by the ubiquitin-editing enzyme A20
  3. Rituximab-mediated Raf kinase inhibitor protein induction modulates NF-κB in Sjögren syndrome
  4. Neovascularization is prominent in the chronic inflammatory lesions of Sjögren's syndrome