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  1. Phosphorylation of USP20 on Ser334 by IRAK-1 Promotes IL-1β-evoked Signaling in Vascular Smooth Muscle Cells and Vascular Inflammation
  2. Drebrin attenuates atherosclerosis by limiting smooth muscle cell transdifferentiation
  3. USP20 (Ubiquitin-Specific Protease 20) Inhibits TNF (Tumor Necrosis Factor)-Triggered Smooth Muscle Cell Inflammation and Attenuates Atherosclerosis
  4. Drebrin regulates angiotensin II-induced aortic remodelling
  5. Interleukin-9 Mediates Chronic Kidney Disease-dependent Vein Graft Disease: a Role for Mast Cells
  6. The Actin-Binding Protein Drebrin Inhibits Neointimal Hyperplasia
  7. Ubiquitin-specific Protease 20 Regulates the Reciprocal Functions of β-Arrestin2 in Toll-like Receptor 4-promoted Nuclear Factor κB (NFκB) Activation
  8. Kruppel-like factor 15 is critical for vascular inflammation
  9. Kalirin Promotes Neointimal Hyperplasia by Activating Rac in Smooth Muscle Cells
  10. Vein graft neointimal hyperplasia is exacerbated by CXCR4 signaling in vein graft-extrinsic cells
  11. G Protein–Coupled Receptor Kinase-5 Attenuates Atherosclerosis by Regulating Receptor Tyrosine Kinases and 7-Transmembrane Receptors
  12. Human Umbilical Cord Blood–Derived Endothelial Cells Reendothelialize Vein Grafts and Prevent Thrombosis
  13. Human umbilical cord blood endothelial progenitor cells decrease vein graft neointimal hyperplasia in SCID mice
  14. Aging-related atherosclerosis is exacerbated by arterial expression of tumor necrosis factor receptor-1: evidence from mouse models and human association studies
  15. Neuropeptide Y Gene Polymorphisms Confer Risk of Early-Onset Atherosclerosis
  16. β-Arrestins Regulate Atherosclerosis and Neointimal Hyperplasia by Controlling Smooth Muscle Cell Proliferation and Migration
  17. Tumor Necrosis Factor Receptor-2 Signaling Attenuates Vein Graft Neointima Formation by Promoting Endothelial Recovery
  18. Expression of Tumor Necrosis Factor Receptor-1 in Arterial Wall Cells Promotes Atherosclerosis
  19. Regulation of the Platelet-derived Growth Factor Receptor-β by G Protein-coupled Receptor Kinase-5 in Vascular Smooth Muscle Cells Involves the Phosphatase Shp2
  20. Activation of vascular smooth muscle cells by TNF and PDGF: overlapping and complementary signal transduction mechanisms
  21. Vein Graft Neointimal Hyperplasia Is Exacerbated by Tumor Necrosis Factor Receptor-1 Signaling in Graft-Intrinsic Cells
  22. Graft-Extrinsic Cells Predominate in Vein Graft Arterialization
  23. A mouse vein graft disease model
  24. Overexpression of G Protein-Coupled Receptor Kinase-2 in Smooth Muscle Cells Reduces Neointimal Hyperplasia