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  1. A primate-specific short GluN2A-NMDA receptor isoform is expressed in the human brain
  2. Inhibition of glycogen synthase kinase-3 by BTA-EG4 reduces tau abnormalities in an organotypic brain slice culture model of Alzheimer’s disease
  3. Membrane association and release of wild-type and pathological tau from organotypic brain slice cultures
  4. Upregulation of calpain activity precedes tau phosphorylation and loss of synaptic proteins in Alzheimer’s disease brain
  5. Astrocytes and neuroinflammation in Alzheimer's disease
  6. Calpain cleavage and inactivation of the sodium calcium exchanger-3 occur downstream of Aβ in Alzheimer's disease