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  1. Monoclonal full-length antibody against TAR DNA-binding protein 43 reduces related proteinopathy in neurons
  2. Mitigation of ALS pathology by neuron-specific inhibition of nuclear factor kappa B signaling
  3. Protective effects of Withania somnifera extract in SOD1G93A mouse model of amyotrophic lateral sclerosis
  4. Recent advances in Japanese encephalitis
  5. Potential Therapeutic Use of Withania somnifera for Treatment of Amyotrophic Lateral Sclerosis
  6. Overview on Japanese Encephalitis in South and Southeast Asia
  7. Withania somnifera Reverses Transactive Response DNA Binding Protein 43 Proteinopathy in a Mouse Model of Amyotrophic Lateral Sclerosis/Frontotemporal Lobar Degeneration
  8. Infections and Inflammation in the Brain and Spinal Cord: A Dangerous Liaison
  9. Ubiquilin-2 drives NF-κB activity and cytosolic TDP-43 aggregation in neuronal cells
  10. Inflammation Induces TDP-43 Mislocalization and Aggregation
  11. TLR7 is a key regulator of innate immunity against Japanese encephalitis virus infection
  12. Role of pattern recognition receptors in flavivirus infections
  13. Neural Stem/Progenitor Cells Induce Conversion of Encephalitogenic T Cells into CD4+-CD25+- FOXP3+ Regulatory T cells
  14. Japanese Encephalitis: A Tale of Inflammation and Degeneration in the Central Nervous System
  15. Histone deacetylase inhibition by Japanese encephalitis virus in monocyte/macrophages: A novel viral immune evasion strategy
  16. Chandipura Virus Induces Neuronal Death through Fas-Mediated Extrinsic Apoptotic Pathway
  17. Japanese encephalitis virus infection modulates the expression of suppressors of cytokine signaling (SOCS) in macrophages: Implications for the hosts’ innate immune response
  18. Japanese encephalitis in India: risk of an epidemic in the National Capital Region
  19. Japanese Encephalitis Virus and Human CNS Infection
  20. Increased resistance of immobilized-stressed mice to infection: Correlation with behavioral alterations
  21. Azithromycin in combination with riboflavin decreases the severity of Staphylococcus aureus infection induced septic arthritis by modulating the production of free radicals and endogenous cytokines
  22. Gentamicin in Combination with Ascorbic Acid Regulates the severity ofStaphylococcus aureusInfection-Induced Septic Arthritis in Mice
  23. STING Mediates Neuronal Innate Immune Response Following Japanese Encephalitis Virus Infection
  24. RIG-I Mediates Innate Immune Response in Mouse Neurons Following Japanese Encephalitis Virus Infection
  25. Chemotherapy In Japanese Encephalitis: Are We There Yet?
  26. Japanese Encephalitis Virus-Infected Macrophages Induce Neuronal Death
  27. Abrogated Inflammatory Response Promotes Neurogenesis in a Murine Model of Japanese Encephalitis
  28. Antiviral and Neuroprotective Role of Octaguanidinium Dendrimer-Conjugated Morpholino Oligomers in Japanese Encephalitis
  29. Minocycline differentially modulates macrophage mediated peripheral immune response following Japanese encephalitis virus infection
  30. Possible Protective Role of Chloramphenicol in TSST-1 and Coagulase-Positive Staphylococcus aureus-Induced Septic Arthritis with Altered Levels of Inflammatory Mediators
  31. Minocycline Differentially Modulates Viral Infection and Persistence in an Experimental Model of Japanese Encephalitis
  32. Neurons under viral attack: Victims or warriors?
  33. A Common Carcinogen Benzo[a]pyrene Causes Neuronal Death in Mouse via Microglial Activation
  34. Understanding the molecular mechanism of blood–brain barrier damage in an experimental model of Japanese encephalitis: Correlation with minocycline administration as a therapeutic agent
  35. Curcumin Protects Neuronal Cells from Japanese Encephalitis Virus-Mediated Cell Death and also Inhibits Infective Viral Particle Formation by Dysregulation of Ubiquitin–Proteasome System
  36. Escherichia coli lipopolysaccharide administration alters antioxidant profile during hypercholesterolemia
  37. Repeated Systemic Escherichia coli Infection Enhances Anti-oxidant Response in Hypercholesterolemic Mice Inducing Cardiovascular Inflammation