All Stories

  1. Chemogenetic Silencing of NaV1.8-Positive Sensory Neurons Reverses Chronic Neuropathic and Bone Cancer Pain in FLEx PSAM4-GlyR Mice

    Article • eNeuro, September 2023, Society for Neuroscience

    Professor John N Wood

  2. Sodium channels

    Article • Brain and Neuroscience Advances, January 2018, SAGE Publications

    Professor John N Wood

  3. From plant extract to molecular panacea: a commentary on Stone (1763) 'An account of the success of the bark of the willow in the cure of the agues'

    Article • Philosophical Transactions of the Royal Society B Biological Sciences, March 2015, Royal Society Publishing

    Professor John N Wood

  4. Pain without Nociceptors? Nav1.7-Independent Pain Mechanisms

    Article • Cell Reports, January 2014, Elsevier

    Professor John N Wood

  5. Novel Mutations Mapping to the Fourth Sodium Channel Domain of Nav1.7 Result in Variable Clinical Manifestations of Primary Erythromelalgia

    Article • NeuroMolecular Medicine, January 2013, Springer Science + Business Media

    Dr Istvan Bodi, Professor John N Wood

  6. Splice Variants of NaV1.7 Sodium Channels Have Distinct β Subunit-Dependent Biophysical Properties

    Article • PLOS One, July 2012, PLOS

    Professor John N Wood

  7. Distinct Nav1.7-dependent pain sensations require different sets of sensory and sympathetic neurons

    Article • Nature Communications, January 2012, Springer Science + Business Media

    Professor John N Wood

  8. Loss-of-function mutations in sodium channel Nav1.7 cause anosmia

    Article • Nature, March 2011, Springer Science + Business Media

    Professor John N Wood

  9. Nociceptor-specific gene deletion reveals a major role for Na v 1.7 (PN1) in acute and inflammatory pain

    Article • Proceedings of the National Academy of Sciences, August 2004, Proceedings of the National Academy of Sciences

    Professor John N Wood