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  1. A transient receptor potential vanilloid 1-dependent corneal–trigeminal neuroinflammatory circuit promotes corneal neuropathy
  2. A transient receptor potential vanilloid 1-dependent corneal-trigeminal neuroinflammatory circuit promotes corneal neuropathy
  3. Aged regulatory T cells fail to control autoimmune lacrimal gland pathogenic CD4+ T cells
  4. Corneal nerves but not corneal epithelial cells are damaged by CD4+ T cells in the ocular surface
  5. Hyaluronate Protects From Benzalkonium Chloride-Induced Ocular Surface Toxicity
  6. Inhibition of Cathepsin S in Autoimmune CD25KO Mouse Improves Sjögren Disease–Like Lacrimal Gland Pathology
  7. Transient Receptor Potential Vanilloid-1 Channels Facilitate Axonal Degeneration of Corneal Sensory Nerves in Dry Eye
  8. CD4+T cells drive corneal nerve damage but not epitheliopathy in an acute aqueous-deficient dry eye model
  9. Regulation of human neutrophil IL-1β secretion induced by Escherichia coli O157:H7 responsible for hemolytic uremic syndrome
  10. Ocular TRPV1 deficiency protects from dry eye-induced corneal nerve damage
  11. Effects of Cathepsin S Inhibition in the Age-Related Dry Eye Phenotype
  12. An ocular Th1 immune response promotes corneal nerve damage independently of the development of corneal epitheliopathy
  13. Ectopic lymphoid structures in the aged lacrimal glands
  14. Molecular mechanisms regulating wound repair: Evidence for paracrine signaling from corneal epithelial cells to fibroblasts and immune cells following transient epithelial cell treatment with Mitomycin C
  15. An ocular Th1 immune response promotes corneal nerve damage independently of the development of corneal epitheliopathy
  16. A, B, C’s of Trk Receptors and Their Ligands in Ocular Repair
  17. Corneal nerves and their role in dry eye pathophysiology
  18. The interplay between serine proteases and caspase-1 regulates the autophagy-mediated secretion of Interleukin-1 beta in human neutrophils
  19. Immune phenotype of the CD4+ T cells in the aged lymphoid organs and lacrimal glands
  20. Age‐related changes in ocular mucosal tolerance: Lessons learned from gut and respiratory tract immunity
  21. The ocular surface immune system through the eyes of aging
  22. Transient tear hyperosmolarity disrupts the neuroimmune homeostasis of the ocular surface and facilitates dry eye onset
  23. Short-Term Fever-Range Hyperthermia Accelerates NETosis and Reduces Pro-inflammatory Cytokine Secretion by Human Neutrophils
  24. The mucosal surfaces of both eyes are immunologically linked by a neurogenic inflammatory reflex involving TRPV1 and substance P
  25. Autophagy Mediates Interleukin-1β Secretion in Human Neutrophils
  26. Mucosal immune tolerance at the ocular surface in health and disease
  27. Mucosal tolerance disruption favors disease progression in an extraorbital lacrimal gland excision model of murine dry eye
  28. Desiccating stress-induced disruption of ocular surface immune tolerance drives dry eye disease
  29. Corneal Asymmetry Analysis by Pentacam Scheimpflug Tomography for Keratoconus Diagnosis
  30. Surface localization of high-mobility group nucleosome-binding protein 2 on leukemic B cells from patients with chronic lymphocytic leukemia is related to secondary autoimmune hemolytic anemia
  31. Multivariate Analysis of the Ocular Response Analyzer’s Corneal Deformation Response Curve for Early Keratoconus Detection
  32. Reply
  33. Restoring Conjunctival Tolerance by Topical Nuclear Factor–κB Inhibitors Reduces Preservative-Facilitated Allergic Conjunctivitis in Mice
  34. Pentacam Scheimpflug Tomography Findings in Topographically Normal Patients and Subclinical Keratoconus Cases
  35. Agreement Between Placido Topography and Scheimpflug Tomography for Corneal Astigmatism Assessment
  36. Combining Ocular Response Analyzer Metrics for Corneal Biomechanical Diagnosis
  37. Benzalkonium chloride breaks down conjunctival immunological tolerance in a murine model
  38. Improved Keratoconus Detection by Ocular Response Analyzer Testing After Consideration of Corneal Thickness as a Confounding Factor
  39. CXCL12-induced chemotaxis is impaired in T cells from patients with ZAP-70-negative chronic lymphocytic leukemia
  40. Chronic Lymphocytic Leukemia Cells Bind and Present the Erythrocyte Protein Band 3: Possible Role as Initiators of Autoimmune Hemolytic Anemia
  41. SHIP‐1 protein level and phosphorylation status differs between CLL cells segregated by ZAP‐70 expression