All Stories

  1. Increased Antiproliferative Activity of Antiestrogens and Neratinib Treatment by Calcitriol in HER2-Positive Breast Cancer Cells
  2. Modulation of the Antitumor Response to Metformin, Caffeine, and Sodium Dichloroacetate by the Hypoxic Microenvironment in Lung Cancer Cells
  3. T-Cell Metabolism and Its Regulation by Checkpoint Molecules: Consequences for Cancer Immunotherapy
  4. Intratumoral Heterogeneity and Metabolic Cross-Feeding in a Three-Dimensional Breast Cancer Culture: An In Silico Perspective
  5. β-D-Glucose-Reduced Silver Nanoparticles Remodel the Tumor Microenvironment in a Murine Model of Triple-Negative Breast Cancer
  6. Metabolic Responses of Lung Adenocarcinoma Cells to Survive under Stressful Conditions Associated with Tumor Microenvironment
  7. The Phytochemical α-Mangostin Inhibits Cervical Cancer Cell Proliferation and Tumor Growth by Downregulating E6/E7-HPV Oncogenes and KCNH1 Gene Expression
  8. Editorial: Molecular basis of epigenetic regulation in cancer therapies
  9. T-Cell Metabolism and Its Regulation by Checkpoint Molecules: Consequences for Cancer Immunotherapy
  10. lncRNA-miRNA-mRNA Interaction Networks Regulation in Hematological Malignancies
  11. The Interplay of GPER1 with 17β-Aminoestrogens in the Regulation of the Proliferation of Cervical and Breast Cancer Cells: A Pharmacological Approach
  12. Antitumoral effects of dovitinib in triple-negative breast cancer are synergized by calcitriol in vivo and in vitro
  13. α-Mangostin Synergizes the Antineoplastic Effects of 5-Fluorouracil Allowing a Significant Dose Reduction in Breast Cancer Cells
  14. Obtaining symmetric and asymmetric bisphosphoramidates and bisphosphoramidothioates by a single step multicomponent reaction
  15. Antitumoral Effects of Dovitinib in Triple-Negative Breast Cancer are Synergized by Calcitriol in Vivo and In Vitro
  16. Determining Factors in the Therapeutic Success of Checkpoint Immunotherapies against PD-L1 in Breast Cancer: A Focus on Epithelial-Mesenchymal Transition Activation
  17. Role of DNA Methylation in the Resistance to Therapy in Solid Tumors
  18. The two-face role of Lactic acidosis in lung cancer
  19. IMMUNEPOTENT CRP plus doxorubicin/cyclophosphamide chemotherapy remodel the tumor microenvironment in an air pouch triple-negative breast cancer murine model
  20. T-Cell Metabolism and Its Dysfunction Induced by Cancer
  21. The Role of Exhaustion in Tumor-Induced T-Cell Dysfunction in Cancer
  22. Synergistic Antitumorigenic Activity of Calcitriol with Curcumin or Resveratrol is Mediated by Angiogenesis Inhibition in Triple Negative Breast Cancer Xenografts
  23. Lactic Acidosis Promotes Mitochondrial Biogenesis in Lung Adenocarcinoma Cells, Supporting Proliferation Under Normoxia or Survival Under Hypoxia
  24. No Sugar Rush for stimulated T cells from lung cancer patients
  25. Astemizole, an Inhibitor of Ether-�-Go-Go-1 Potassium Channel, Increases the Activity of the Tyrosine Kinase Inhibitor Gefitinib in Breast Cancer Cells
  26. Mitochondrial calcium: Transport and modulation of cellular processes in homeostasis and cancer (Review)
  27. Editorial: Immuno-Metabolism in Tumor Microenvironment
  28. Lung tumor cells restrain T cells, the PD-L1/PD-1 checkpoint inhibitory pathway
  29. Lactate Contribution to the Tumor Microenvironment: Mechanisms, Effects on Immune Cells and Therapeutic Relevance
  30. In VivoChemoprotective Activity of Bovine Dialyzable Leukocyte Extract in Mouse Bone Marrow Cells against Damage Induced by 5-Fluorouracil
  31. 529 LAP TGF-beta subset of CD4+CD25+CD127- Treg cells is increased and overexpresses LAP TGF-beta in lung adenocarcinoma patients
  32. Calcitriol and its analogues enhance the antiproliferative activity of gefitinib in breast cancer cells
  33. Antihormonal agents as a strategy to improve the effect of chemo-radiation in cervical cancer: in vitro and in vivo study
  34. LAP TGF-Beta Subset of CD4+CD25+CD127−Treg Cells is Increased and Overexpresses LAP TGF-Beta in Lung Adenocarcinoma Patients
  35. The Role of Exhaustion in Tumor-Induced T Cell Dysfunction in Cancer
  36. Relationship of Dendritic Cell Density, HMGB1 Expression, and Tumor-infiltrating Lymphocytes in Non–Small Cell Lung Carcinomas
  37. Activated T Cells, but Not Lung Tumor Cells, Express Fas Ligand
  38. Transcriptional analysis of hnRNPA0, A1, A2, B1, and A3 in lung cancer cell lines in response to acidosis, hypoxia, and serum deprivation conditions
  39. Tumor-Induced CD8+ T-Cell Dysfunction in Lung Cancer Patients
  40. Activation-induced cell death of memory CD8+ T cells from pleural effusion of lung cancer patients is mediated by the type II Fas-induced apoptotic pathway
  41. Tumor cell metabolism
  42. Lung carcinomas do not induce T-cell apoptosis via the Fas/Fas ligand pathway but down-regulate CD3 epsilon expression
  43. Lung carcinomas decrease the number of monocytes/macrophages (CD14+ cells) that produce TNF-α
  44. Lung squamous cell carcinoma and adenocarcinoma cell lines use different mediators to induce comparable phenotypic and functional changes in human monocyte-derived dendritic cells
  45. Effector, memory and naïve CD8+ T cells in peripheral blood and pleural effusion from lung adenocarcinoma patients
  46. Apoptosis and cell cycle disturbances induced by coumarin and 7-hydroxycoumarin on human lung carcinoma cell lines
  47. Lack of correlation between growth inhibition by TGF-β and the percentage of cells expressing type II TGF-β receptor in human non-small cell lung carcinoma cell lines