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  1. Data from Depleting IL1R2<sup>+</sup> Tumor-Infiltrating Regulatory T Cells with an ADCC-Prone Nanobody Construct Boosts the Efficacy of Anti–PD-1 Immunotherapy
  2. Figure S1 from Depleting IL1R2<sup>+</sup> Tumor-Infiltrating Regulatory T Cells with an ADCC-Prone Nanobody Construct Boosts the Efficacy of Anti–PD-1 Immunotherapy
  3. Figure S10 from Depleting IL1R2<sup>+</sup> Tumor-Infiltrating Regulatory T Cells with an ADCC-Prone Nanobody Construct Boosts the Efficacy of Anti–PD-1 Immunotherapy
  4. Figure S11 from Depleting IL1R2<sup>+</sup> Tumor-Infiltrating Regulatory T Cells with an ADCC-Prone Nanobody Construct Boosts the Efficacy of Anti–PD-1 Immunotherapy
  5. Figure S12 from Depleting IL1R2<sup>+</sup> Tumor-Infiltrating Regulatory T Cells with an ADCC-Prone Nanobody Construct Boosts the Efficacy of Anti–PD-1 Immunotherapy
  6. Figure S13 from Depleting IL1R2<sup>+</sup> Tumor-Infiltrating Regulatory T Cells with an ADCC-Prone Nanobody Construct Boosts the Efficacy of Anti–PD-1 Immunotherapy
  7. Figure S14 from Depleting IL1R2<sup>+</sup> Tumor-Infiltrating Regulatory T Cells with an ADCC-Prone Nanobody Construct Boosts the Efficacy of Anti–PD-1 Immunotherapy
  8. Figure S15 from Depleting IL1R2<sup>+</sup> Tumor-Infiltrating Regulatory T Cells with an ADCC-Prone Nanobody Construct Boosts the Efficacy of Anti–PD-1 Immunotherapy
  9. Figure S16 from Depleting IL1R2<sup>+</sup> Tumor-Infiltrating Regulatory T Cells with an ADCC-Prone Nanobody Construct Boosts the Efficacy of Anti–PD-1 Immunotherapy
  10. Figure S2 from Depleting IL1R2<sup>+</sup> Tumor-Infiltrating Regulatory T Cells with an ADCC-Prone Nanobody Construct Boosts the Efficacy of Anti–PD-1 Immunotherapy
  11. Figure S3 from Depleting IL1R2<sup>+</sup> Tumor-Infiltrating Regulatory T Cells with an ADCC-Prone Nanobody Construct Boosts the Efficacy of Anti–PD-1 Immunotherapy
  12. Figure S4 from Depleting IL1R2<sup>+</sup> Tumor-Infiltrating Regulatory T Cells with an ADCC-Prone Nanobody Construct Boosts the Efficacy of Anti–PD-1 Immunotherapy
  13. Figure S5 from Depleting IL1R2<sup>+</sup> Tumor-Infiltrating Regulatory T Cells with an ADCC-Prone Nanobody Construct Boosts the Efficacy of Anti–PD-1 Immunotherapy
  14. Figure S6 from Depleting IL1R2<sup>+</sup> Tumor-Infiltrating Regulatory T Cells with an ADCC-Prone Nanobody Construct Boosts the Efficacy of Anti–PD-1 Immunotherapy
  15. Figure S7 from Depleting IL1R2<sup>+</sup> Tumor-Infiltrating Regulatory T Cells with an ADCC-Prone Nanobody Construct Boosts the Efficacy of Anti–PD-1 Immunotherapy
  16. Figure S8 from Depleting IL1R2<sup>+</sup> Tumor-Infiltrating Regulatory T Cells with an ADCC-Prone Nanobody Construct Boosts the Efficacy of Anti–PD-1 Immunotherapy
  17. Figure S9 from Depleting IL1R2<sup>+</sup> Tumor-Infiltrating Regulatory T Cells with an ADCC-Prone Nanobody Construct Boosts the Efficacy of Anti–PD-1 Immunotherapy
  18. Depleting IL1R2+ Tumor-Infiltrating Regulatory T Cells with an ADCC-Prone Nanobody Construct Boosts the Efficacy of Anti-PD1 Immunotherapy
  19. Imaging of tumor-associated macrophage dynamics during immunotherapy using a CD163-specific nanobody-based immunotracer
  20. Exploring the Endocrine Mechanisms in Adenomyosis: From Pathogenesis to Therapies
  21. Exploring the Endocrine Mechanisms in Adenomyosis: From Pathogenesis to Therapies
  22. Dietary supplementation of cystinotic mice by lysine inhibits the megalin pathway and decreases kidney cystine content
  23. Reviewers of the Year 2022: F&S Science celebrates excellence in our world class reviewers
  24. Quantification of lung fibrosis in COVID-19 related and -unrelated ARDS
  25. Cover Image, Volume 59, Issue 7‐8
  26. Semi‐automated digital quantification of cellular infiltrates for in vivo evaluation of transplanted islets of Langerhans encapsulated with bioactive materials
  27. Generation and characterization of a tamoxifen‐inducible Vsx1‐CreERT2 line to target V2 interneurons in the mouse developing spinal cord
  28. Digital Image Analysis of Picrosirius Red Staining: A Robust Method for Multi-Organ Fibrosis Quantification and Characterization
  29. Adipose tissue–derived stem cells protect the primordial follicle pool from both direct follicle death and abnormal activation after ovarian tissue transplantation
  30. Canagliflozin protects the vascular barrier during sepsis by AMPK dependant mechanisms
  31. α1AMP-Activated Protein Kinase Protects against Lipopolysaccharide-Induced Endothelial Barrier Disruption via Junctional Reinforcement and Activation of the p38 MAPK/HSP27 Pathway
  32. Accurate and live peroxisome biogenesis evaluation achieved by lentiviral expression of a green fluorescent protein fused to a peroxisome targeting signal 1
  33. Fibroid management in premenopausal women
  34. Molecular mechanisms responsible for myoma volume reduction after ulipristal acetate
  35. Ulipristal acetate for the management of large uterine fibroids associated with heavy bleeding: a review
  36. Gene expression changes in uterine myomas in response to ulipristal acetate treatment
  37. Matrix Metalloproteinase Activity Correlates With Uterine Myoma Volume Reduction After Ulipristal Acetate Treatment
  38. Progesterone Receptor Isoforms, Nuclear Corepressor-1 and Steroid Receptor Coactivator-1 and B-Cell Lymphoma 2 and Akt and Akt Phosphorylation Status in Uterine Myomas after Ulipristal Acetate Treatment: A Systematic Immunohistochemical Evaluation
  39. In vivo mechanisms of uterine myoma volume reduction with ulipristal acetate treatment