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  1. Inhibition of inflammation by minocycline improves heart failure and depression-like behaviour in rats after myocardial infarction
  2. Central and Peripheral Slow-Pressor Mechanisms Contributing to
  3. Update on angiotensin II: new endocrine connections between the brain, adrenal glands and the cardiovascular system
  4. Enhanced expression of epithelial sodium channels causes salt-induced hypertension in mice through inhibition of the α2 -isoform of Na+ , K+ -ATPase
  5. Knockdown of mineralocorticoid or angiotensin II type 1 receptor gene expression in the paraventricular nucleus prevents angiotensin II hypertension in rats
  6. Mineralocorticoid and AT1 receptors in the paraventricular nucleus contribute to sympathetic hyperactivity and cardiac dysfunction in rats post myocardial infarct