All Stories

  1. Early‐life and chronic exposure to high‐fat diet alters noradrenergic and glutamatergic neurotransmission in the male rat amygdala and hippocampus under cognitive challenges
  2. Paradoxical Boosting of Weak and Strong Spatial Memories by Hippocampal Dopamine Uncaging
  3. The anterior insula and its projection to amygdala nuclei modulate the abstinence-exacerbated expression of conditioned place preference
  4. Synergistic photoactivation of VTA-catecholaminergic and BLA-glutamatergic projections induces long-term potentiation in the insular cortex
  5. Top-down circuitry from the anterior insular cortex to VTA dopamine neurons modulates reward-related memory
  6. Salience to remember: VTA-IC dopaminergic pathway activity is necessary for object recognition memory formation
  7. Inhibition of hippocampal palmitoyl acyltransferase activity impairs spatial learning and memory consolidation
  8. Transforming experiences: Neurobiology of memory updating/editing
  9. Spatial contextual recognition memory updating is modulated by dopamine release in the dorsal hippocampus from the locus coeruleus
  10. Voluntary physical activity improves spatial and recognition memory deficits induced by post-weaning chronic exposure to a high-fat diet
  11. Cognitive Impairment in Alzheimer’s and Metabolic Diseases: A Catecholaminergic Hypothesis
  12. Dopamine activity on the perceptual salience for recognition memory
  13. Photostimulation of Ventral Tegmental Area-Insular Cortex Dopaminergic Inputs Enhances the Salience to Consolidate Aversive Taste Recognition Memory via D1-Like Receptors
  14. Photostimulation of VTA-IC dopaminergic inputs enhances the salience to consolidate aversive taste recognition memory via D1 receptors
  15. Maintenance of conditioned place avoidance induced by gastric malaise requires NMDA activity within the ventral hippocampus
  16. Catecholaminergic stimulation restores high-sucrose diet-induced hippocampal dysfunction
  17. Cortical neurochemical signaling of gustatory stimuli and their visceral consequences during the acquisition and consolidation of taste aversion memory
  18. Class I HDAC inhibition improves object recognition memory consolidation through BDNF/TrkB pathway in a time-dependent manner
  19. Transcriptional, Behavioral and Biochemical Profiling in the 3xTg-AD Mouse Model Reveals a Specific Signature of Amyloid Deposition and Functional Decline in Alzheimer’s Disease
  20. Age-Dependent Decline in Synaptic Mitochondrial Function Is Exacerbated in Vulnerable Brain Regions of Female 3xTg-AD Mice
  21. Telomere length and oxidative stress variations in a murine model of Alzheimer’s disease progression
  22. Glutamatergic basolateral amygdala to anterior insular cortex circuitry maintains rewarding contextual memory
  23. Artificial taste avoidance memory induced by coactivation of NMDA and β-adrenergic receptors in the amygdala
  24. Telomere length and oxidative stress variations in a murine model of Alzheimer’s disease progression
  25. Early memory consolidation window enables drug induced state-dependent memory
  26. Plasticity in The Central Nervous System
  27. Differential requirement of de novo Arc protein synthesis in the insular cortex and the amygdala for safe and aversive taste long-term memory formation
  28. Object Recognition and Object Location Recognition Memory – The Role of Dopamine and Noradrenaline
  29. Recurrent moderate hypoglycemia exacerbates oxidative damage and neuronal death leading to cognitive dysfunction after the hypoglycemic coma
  30. Determinants to trigger memory reconsolidation: The role of retrieval and updating information
  31. Memory reconsolidation and memory updating: Two sides of the same coin?
  32. Hippocampal release of dopamine and norepinephrine encodes novel contextual information
  33. Memory trace reactivation and behavioral response during retrieval are differentially modulated by amygdalar glutamate receptors activity: interaction between amygdala and insular cortex
  34. Decreased levels of NMDA but not AMPA receptors in the lipid-raft fraction of 3xTg-AD model of Alzheimer's disease: Relation to Arc/Arg3.1 protein expression
  35. Corrigendum to “Differential involvement of glutamatergic and catecholaminergic activity within the amygdala during taste aversion retrieval on memory expression and updating” [Behav. Brain Res. 307 (2016) 120–125]
  36. Differential involvement of glutamatergic and catecholaminergic activity within the amygdala during taste aversion retrieval on memory expression and updating
  37. Dopaminergic neurotransmission dysfunction induced by amyloid-β transforms cortical long-term potentiation into long-term depression and produces memory impairment
  38. Neural ablation of the PARK10 candidate Plpp3 leads to dopaminergic transmission deficits without neurodegeneration
  39. Spatial Memory Impairment is Associated with Intraneural Amyloid-β Immunoreactivity and Dysfunctional Arc Expression in the Hippocampal-CA3 Region of a Transgenic Mouse Model of Alzheimer’s Disease
  40. Consolidation and reconsolidation of object recognition memory
  41. Effects of glutamate and its metabotropic receptors class 1 antagonist in appetitive taste memory formation
  42. Identification of age- and disease-related alterations in circulating miRNAs in a mouse model of Alzheimer's disease
  43. New Insights on Retrieval-Induced and Ongoing Memory Consolidation: Lessons from Arc
  44. Differential role of insular cortex muscarinic and NMDA receptors in one-trial appetitive taste learning
  45. Retrieval is not necessary to trigger reconsolidation of object recognition memory in the perirhinal cortex
  46. Age-Dependent Increment of Hydroxymethylation in the Brain Cortex in the Triple-Transgenic Mouse Model of Alzheimer's Disease
  47. Role of glutamate receptors of central and basolateral amygdala nuclei on retrieval and reconsolidation of taste aversive memory
  48. The forgotten insular cortex: Its role on recognition memory formation
  49. Retrieval and reconsolidation of object recognition memory are independent processes in the perirhinal cortex
  50. Dopamine D1 receptor activity modulates object recognition memory consolidation in the perirhinal cortex but not in the hippocampus
  51. Taste aversion memory reconsolidation is independent of its retrieval
  52. Restoration of dopamine release deficits during object recognition memory acquisition attenuates cognitive impairment in a triple transgenic mice model of Alzheimer's disease
  53. Post-acquisition release of glutamate and norepinephrine in the amygdala is involved in taste-aversion memory consolidation
  54. Muscarinic receptors activity in the perirhinal cortex and hippocampus has differential involvement in the formation of recognition memory
  55. Interplay of amygdala and insular cortex during and after associative taste aversion memory formation
  56. Brain-immune interactions and the neural basis of disease-avoidant ingestive behaviour
  57. Caspase-12 Activation is Involved in Amyloid-β Protein-Induced Synaptic Toxicity
  58. Familiar taste induces higher dendritic levels of activity-regulated cytoskeleton-associated protein in the insular cortex than a novel one
  59. Long-term aversive taste memory requires insular and amygdala protein degradation
  60. Post-learning molecular reactivation underlies taste memory consolidation
  61. Erratum
  62. Differential participation of temporal structures in the consolidation and reconsolidation of taste aversion extinction
  63. Off‐line concomitant release of dopamine and glutamate involvement in taste memory consolidation
  64. Is memory consolidation a multiple-circuit system?
  65. Simultaneous but not independent anisomycin infusions in insular cortex and amygdala hinder stabilization of taste memory when updated
  66. Safe taste memory consolidation is disrupted by a protein synthesis inhibitor in the nucleus accumbens shell
  67. Medial temporal lobe structures participate differentially in consolidation of safe and aversive taste memories
  68. The consolidation of object and context recognition memory involve different regions of the temporal lobe
  69. Spatial memory formation induces recruitment of NMDA receptor and PSD‐95 to synaptic lipid rafts
  70. Taste novelty induces intracellular redistribution of NR2A and NR2B subunits of NMDA receptor in the insular cortex
  71. Intrahippocampal anisomycin infusions disrupt previously consolidated spatial memory only when memory is updated
  72. Remodeling of hippocampal mossy fibers is selectively induced seven days after the acquisition of a spatial but not a cued reference memory task
  73. PKC blockade differentially affects aversive but not appetitive gustatory memories
  74. Activation of hippocampal postsynaptic muscarinic receptors is involved in long‐term spatial memory formation
  75. Cholinergic activity in the insular cortex is necessary for acquisition and consolidation of contextual memory
  76. The expression of TRH, its receptors and degrading enzyme is differentially modulated in the rat limbic system during training in the Morris water maze
  77. Taste Memory Formation: Role of Nucleus Accumbens
  78. Reply to 'Physician, heal thyself'
  79. NMDA and muscarinic receptors of the nucleus accumbens have differential effects on taste memory formation
  80. Basolateral amygdala glutamatergic activation enhances taste aversion through NMDA receptor activation in the insular cortex
  81. Insular cortex is involved in consolidation of object recognition memory
  82. Protein synthesis underlies post-retrieval memory consolidation to a restricted degree only when updated information is obtained
  83. Analysis of the Stress Response in Rats Trained in the Water-Maze: Differential Expression of Corticotropin-Releasing Hormone, CRH-R1, Glucocorticoid Receptors and Brain-Derived Neurotrophic Factor in Limbic Regions
  84. Neurobiology of Taste-recognition Memory Formation
  85. Cognitive deficits in adult rats by lead intoxication are related with regional specific inhibition of cNOS
  86. Molecular Signals into the Insular Cortex and Amygdala During Aversive Gustatory Memory Formation
  87. Enhancement of antibody response by one-trial conditioning: Contrasting results using different antigens
  88. Perirhinal Cortex Muscarinic Receptor Blockade Impairs Taste Recognition Memory Formation
  89. Blockade of noradrenergic receptors in the basolateral amygdala impairs taste memory
  90. Cholinergic dependence of taste memory formation: Evidence of two distinct processes
  91. Role of cholinergic system on the construction of memories: Taste memory encoding
  92. Blockade of cortical muscarinic but not NMDA receptors prevents a novel taste from becoming familiar
  93. The role of cortical cholinergic pre- and post-synaptic receptors in taste memory formation
  94. Peripheral protein immunization induces rapid activation of the CNS, as measured by c-Fos expression
  95. Differential involvement of cortical muscarinic and NMDA receptors in short‐ and long‐term taste aversion memory
  96. Glutamatergic activity in the amygdala signals visceral input during taste memory formation
  97. Differential effects of bicuculline and muscimol microinjections into the nucleus basalis magnocellularis in taste and place aversive memory formation
  98. In vivo effects of intracortical administration of NMDA and metabotropic glutamate receptors antagonists on neocortical long-term potentiation and conditioned taste aversion
  99. Cortical cholinergic activity is related to the novelty of the stimulus
  100. Differential participation of the NBM in the acquisition and retrieval of conditioned taste aversion and Morris water maze
  101. Long-term potentiation in the insular cortex enhances conditioned taste aversion retention
  102. Differential effects of 192IgG-saporin and NMDA-induced lesions into the basal forebrain on cholinergic activity and taste aversion memory formation
  103. Reversible inactivation of the nucleus basalis magnocellularis induces disruption of cortical acetylcholine release and acquisition, but not retrieval, of aversive memories
  104. Blockade of N-methyl-d-aspartate receptors in the insular cortex disrupts taste aversion and spatial memory formation
  105. Conditioned Enhancement of Antibody Production Is Disrupted by Insular Cortex and Amygdala but Not Hippocampal Lesions
  106. Synaptogenesis of mossy fibers induced by spatial water maze overtraining
  107. Differential Effects of NMDA-Induced Lesions into the Insular Cortex and Amygdala on the Acquisition and Evocation of Conditioned Immunosuppression
  108. Acetylcholine determination of microdialysates of fetal neocortex grafts that induce recovery of learning
  109. In vivo long-term potentiation in the insular cortex: NMDA receptor dependence
  110. Long-term memory retrieval deficits of learned taste aversions are ameliorated by cortical fetal brain implants.
  111. Recovery of taste aversion learning induced by fetal neocortex grafts: correlation with in vivo extracellular acetylcholine
  112. Insular Cortex and Amygdala Lesions Induced after Aversive Training Impair Retention: Effects of Degree of Training
  113. Differential Effects of Anterior and Posterior Insular Cortex Lesions on the Acquisition of Conditioned Taste Aversion and Spatial Learning
  114. Insular Cortex Lesions Impair the Acquisition of Conditioned Immunosuppression
  115. Morphometric study of fetal brain transplants in the insular cortex and NGF effects on neuronal and glial development
  116. Enhancement of Antibody Production by a Learning Paradigm
  117. Differential recovery of inhibitory avoidance learning by striatal, cortical, and mesencephalic fetal grafts
  118. Accelerating behavioral recovery after cortical lesions
  119. Accelerating behavioral recovery after cortical lesions
  120. Graft-induced Recovery of Inhibitory Avoidance Conditioning in Striatal Lesioned Rats is Related to Choline Acetyltransferase Activity
  121. Effects of catecholaminergic depletion of the amygdala and insular cortex on the potentiation of odor by taste aversions
  122. Hypothalamic but not cortical grafts induce recovery of sexual behavior and connectivity in medial preoptic area-lesioned rats
  123. Effects of excitotoxic lesions of the nucleus basalis magnocellularis on conditioned taste aversion and inhibitory avoidance in the rat
  124. Adrenal Medullary Grafts Restore Olfactory Deficits and Catecholamine Levels of 6-OHDA Amygdala Lesioned Animals
  125. Nerve Growth Factor with Insular Cortical Grafts Induces Recovery of Learning and Reestablishes Graft Choline Acetyltransferase Activity
  126. Insular Cortical Grafts: Factors Affecting the Recovery of Learning
  127. Insular cortex and amygdala lesions differentially affect acquisition on inhibitory avoidance and conditioned taste aversion
  128. Time-dependent recovery of taste aversion learning by fetal brain transplants in gustatory neocortex-lesioned rats
  129. Correlation between acetylcholine release and recovery of conditioned taste aversion induced by fetal neocortex grafts
  130. Fetal brain transplants induce recovery of male sexual behavior in medial preoptic area-lesioned rats
  131. Release of acetylcholine, γ-aminobutyrate, dopamine and glutamate, and activity of some related enzymes, in rat gustatory neocortex
  132. The conditioned stimulus-unconditioned stimulus-feedback feeding sequence: Reply to Ellins, von Kluge, and Cramer (1990).
  133. Fetal brain grafts induce recovery of learning deficits and connectivity in rats with gustatory neocortex lesion
  134. Odor and taste aversions conditioned in anesthetized rats.
  135. Taste-potentiated noise-illness associations.
  136. Fetal brain transplants induce recuperation of taste aversion learning
  137. Potentiation of odor by taste and odor aversions in rats are regulated by cholinergic activity of dorsal hippocampus
  138. Flavor-illness aversions: The role of the amygdala in the acquisition of taste-potentiated odor aversions
  139. Is cholinergic activity of the striatum involved in the acquisition of positively-motivated behaviors?
  140. A General Theory of Aversion Learninga
  141. Fetal suprachiasmatic nucleus transplants: diurnal rhythm recovery of lesioned rats
  142. Addictive agents and intracranial stimulation (ICS): Novel antagonists and agonists of morphine and pressing for ICS
  143. Flavor—illness aversions: Potentiation of odor by taste is disrupted by application of novocaine into amygdala
  144. Differential [35S]methionine incorporation into protein of different brain areas of the rat during a learning task
  145. Intracerebral administration of naloxone and drinking in water-deprived rats
  146. Cholinergic blockade of the caudate nucleus and spatial alternation performance in rats: Overtraining induced protection against behavioral deficits