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  1. Impaired Dopamine Signaling in Early Diabetic Retina: Insights from D1R and D4R Agonist Effects on Whole Retina Responses
  2. Visual Dysfunction in Diabetes
  3. Impaired Light Adaptation of ON-Sustained Ganglion Cells in Early Diabetes Is Attributable to Diminished Response to Dopamine D4 Receptor Activation
  4. Dopamine D1 and D4 receptors contribute to light adaptation in ON-sustained retinal ganglion cells
  5. Impaired light adaptation of ON-sustained ganglion cells in early diabetes is attributable to diminished dopamine D4 receptor sensitivity
  6. Early diabetes impairs ON sustained ganglion cell light responses and adaptation without cell death or dopamine insensitivity
  7. Dopamine D1 and D4 receptors contribute to light adaptation in ON-sustained retinal ganglion cells
  8. Inhibitory components of retinal bipolar cell receptive fields are differentially modulated by dopamine D1 receptors
  9. The effects of early diabetes on inner retinal neurons
  10. Reductions in Calcium Signaling Limit Inhibition to Diabetic Retinal Rod Bipolar Cells
  11. Dopamine D1 receptor activation reduces local inner retinal inhibition to light-adapted levels
  12. Dopamine D1 receptor activation contributes to light-adapted changes in retinal inhibition to rod bipolar cells
  13. Early Retinal Neuronal Dysfunction in Diabetic Mice: Reduced Light-Evoked Inhibition Increases Rod Pathway Signaling
  14. Light adaptation alters inner retinal inhibition to shape OFF retinal pathway signaling
  15. Different types of retinal inhibition have distinct neurotransmitter release properties
  16. Light adaptation alters the source of inhibition to the mouse retinal OFF pathway
  17. Slow changes in Ca2+ cause prolonged release from GABAergic retinal amacrine cells
  18. Inhibition to retinal rod bipolar cells is regulated by light levels
  19. Nonlinear Interactions between Excitatory and Inhibitory Retinal Synapses Control Visual Output
  20. Rod Vision Is Controlled by Dopamine-Dependent Sensitization of Rod Bipolar Cells by GABA
  21. Signal Processing in the Inner Retina
  22. Multiple pathways of inhibition shape bipolar cell responses in the retina
  23. Interneuron Circuits Tune Inhibition in Retinal Bipolar Cells
  24. Development of Presynaptic Inhibition Onto Retinal Bipolar Cell Axon Terminals Is Subclass-Specific
  25. Visual Signal Processing in the Inner Retina
  26. Presynaptic inhibition differentially shapes transmission in distinct circuits in the mouse retina
  27. Receptor and Transmitter Release Properties Set the Time Course of Retinal Inhibition
  28. GABAA, GABACand glycine receptor-mediated inhibition differentially affects light-evoked signalling from mouse retinal rod bipolar cells
  29. GABAC receptor-mediated inhibition in the retina
  30. Mechanisms for the Modulation of Native Glycine Receptor Channels by Ethanol
  31. Differential Effects of Ethanol on GABAA and Glycine Receptor-Mediated Synaptic Currents in Brain Stem Motoneurons