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  1. Hypoglycaemic stimulation of macrophage cytokine release is suppressed by AMP‐activated protein kinase activation
  2. Validation of a refined protocol for mouse oral glucose tolerance testing without gavage
  3. Regulation of astrocyte metabolism by mitochondrial translocator protein 18 kDa
  4. Pharmacological AMP-activated protein kinase activation suppresses low glucose-dependent macrophage migration inhibitory factor release from macrophages
  5. Regulation of astrocyte metabolism by mitochondrial translocator protein 18kDa
  6. Amino acid homeostasis is a target of metformin therapy
  7. Impact of chemogenetic activation of dorsal vagal complex astrocytes in mice on adaptive glucoregulatory responses
  8. Glutamate Prevents Altered Mitochondrial Function Following Recurrent Low Glucose in Hypothalamic but Not Cortical Primary Rat Astrocytes
  9. Brain Permeable AMP-Activated Protein Kinase Activator R481 Raises Glycaemia by Autonomic Nervous System Activation and Amplifies the Counterregulatory Response to Hypoglycaemia in Rats
  10. Astrocytes in the dorsal vagal complex are not activated by systemic glucoprivation and their chemogenetic activation does not elicit homeostatic glucoregulatory responses in mice
  11. Absence of the mitochondrial translocator protein 18 kDa in mice does not affect body weight or food intake responses to altered energy availability
  12. Attenuated Induction of the Unfolded Protein Response in Adult Human Primary Astrocytes in Response to Recurrent Low Glucose
  13. Brain-Body Control of Glucose Homeostasis—Insights From Model Organisms
  14. Extracellular ATP Increases Glucose Metabolism in Skeletal Muscle Cells in a P2 Receptor Dependent Manner but Does Not Contribute to Palmitate-Induced Insulin Resistance
  15. Analysis of the transcriptome and DNA methylome in response to acute and recurrent low glucose in human primary astrocytes
  16. The metabolic response to inflammation in astrocytes is regulated by nuclear factor‐kappa B signaling
  17. Changes in neuronal activity across the mouse ventromedial nucleus of the hypothalamus in response to low glucose: evaluation using an extracellular multi‐electrode array approach
  18. Regulation of food intake by astrocytes in the brainstem dorsal vagal complex
  19. Immunometabolic Changes in Glia – A Potential Role in the Pathophysiology of Obesity and Diabetes
  20. Brain permeable AMPK activator R481 raises glycemia by autonomic nervous system activation and amplifies the counterregulatory response to hypoglycemia in rats
  21. Astrocytes in neuroendocrine systems: An overview
  22. Basal fatty acid oxidation increases after recurrent low glucose in human primary astrocytes
  23. Regulation of hepatic glucose production and AMPK by AICAR but not by metformin depends on drug uptake through the equilibrative nucleoside transporter 1 (ENT1)
  24. Human primary astrocytes increase basal fatty acid oxidation following recurrent low glucose to maintain intracellular nucleotide levels
  25. The species origin of the cellular microenvironment influences markers of beta cell fate and function in EndoC-βH1 cells
  26. CNS Targets of Adipokines
  27. AMP-activated protein kinase (AMPK) activator A-769662 increases intracellular calcium and ATP release from astrocytes in an AMPK-independent manner
  28. Oleate induces K ATP channel-dependent hyperpolarization in mouse hypothalamic glucose-excited neurons without altering cellular energy charge
  29. Chronic exposure to KATP channel openers results in attenuated glucose sensing in hypothalamic GT1-7 neurons
  30. Anti-Inflammatory Effects of Metformin Irrespective of Diabetes Status
  31. Chronic Activation of γ2 AMPK Induces Obesity and Reduces β Cell Function
  32. Anti-inflammatory effects of metformin and their relationship to the therapeutic action of the drug
  33. Characterisation of the astrocytic response to acute and recurrent hypoglycaemia
  34. Kv1.3 inhibitors have differential effects on glucose uptake and AMPK activity in skeletal muscle cell lines and mouse ex vivo skeletal muscle
  35. Continuous hypothalamic KATP activation blunts glucose counter-regulation in vivo in rats and suppresses KATP conductance in vitro
  36. AMPK modulates glucose-sensing in insulin-secreting cells by altered phosphotransfer to KATP channels
  37. Hypoglycaemia: Exercise for the Brain?
  38. Mouse hypothalamic GT1-7 cells demonstrate AMPK-dependent intrinsic glucose-sensing behaviour
  39. The physiology and pathophysiology of the neural control of the counterregulatory response
  40. Loss of AMP-activated protein kinase α2 subunit in mouse β-cells impairs glucose-stimulated insulin secretion and inhibits their sensitivity to hypoglycaemia