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  1. iNOS inhibition reduces alterations induced by ROFA (particulate matter).
  2. The Plant Proteinase Inhibitor CrataBL Plays a Role in Controlling Asthma Response in Mice
  3. A Plant Proteinase Inhibitor from Enterolobium contortisiliquum Attenuates Pulmonary Mechanics, Inflammation and Remodeling Induced by Elastase in Mice
  4. Prophylactic and therapeutic treatment with the flavonone sakuranetin ameliorates LPS-induced acute lung injury
  5. Acute lung injury is reduced by the α7nAChR agonist
  6. Plant Proteinase Inhibitor BbCI Modulates Lung Inflammatory Responses and Mechanic and Remodeling Alterations Induced by Elastase in Mice
  7. Structurally Related Monoterpenes p-Cymene, Carvacrol and Thymol Isolated from Essential Oil from Leaves of Lippia sidoides Cham. (Verbenaceae) Protect Mice against Elastase-Induced Emphysema
  8. Reduced expression of VAChT increases renal fibrosis
  9. Sakuranetin reverses vascular peribronchial and lung parenchyma remodeling in a murine model of chronic allergic pulmonary inflammation
  10. The Plant-DerivedBauhinia bauhinioidesKallikrein Proteinase Inhibitor (rBbKI) Attenuates Elastase-Induced Emphysema in Mice
  11. The Role of Acetylcholine in the Inflammatory Response in Animals Surviving Sepsis Induced by Cecal Ligation and Puncture
  12. A plant proteinase inhibitor from Crataeva tapia (CrataBL) attenuates elastase-induced pulmonary inflammatory, remodeling, and mechanical alterations in mice
  13. Low level laser therapy reduces acute lung inflammation without impairing lung function
  14. Erratum: A flavanone from Baccharis retusa (Asteraceae) prevents elastase-induced emphysema in mice by regulating NF-κB, oxidative stress and metalloproteinases
  15. Y-27632 is associated with corticosteroid-potentiated control of pulmonary remodeling and inflammation in guinea pigs with chronic allergic inflammation
  16. A flavanone from Baccharis retusa (Asteraceae) prevents elastase-induced emphysema in mice by regulating NF-κB, oxidative stress and metalloproteinases
  17. Increased Airway Reactivity and Hyperinsulinemia in Obese Mice Are Linked by ERK Signaling in Brain Stem Cholinergic Neurons
  18. Pulmonary Inflammation Is Regulated by the Levels of the Vesicular Acetylcholine Transporter
  19. A Treatment with a Protease Inhibitor Recombinant from the Cattle Tick (Rhipicephalus Boophilus microplus) Ameliorates Emphysema in Mice
  20. Structure-Activity Association of Flavonoids in Lung Diseases
  21. Effects of Rho-kinase inhibition in lung tissue with chronic inflammation
  22. Modulation of the oscillatory mechanics of lung tissue and the oxidative stress response induced by arginase inhibition in a chronic allergic inflammation model
  23. Flavonone treatment reverses airway inflammation and remodelling in an asthma murine model
  24. Eosinophilic Inflammation in Allergic Asthma
  25. Antileukotriene Reverts the Early Effects of Inflammatory Response of Distal Parenchyma in Experimental Chronic Allergic Inflammation
  26. Effects of corticosteroid, montelukast and iNOS inhibition on distal lung with chronic inflammation
  27. Rho-kinase inhibition attenuates airway responsiveness, inflammation, matrix remodeling, and oxidative stress activation induced by chronic inflammation
  28. Stress amplifies lung tissue mechanics, inflammation and oxidative stress induced by chronic inflammation
  29. Reduction Of Cholinergic Neurotransmission Causes Lung Mechanics Alterations Associated To Airway Inflammation And Remodeling In Mice
  30. Inducible Nitric Oxide Synthase Inhibition Attenuates Physical Stress-Induced Lung Hyper-Responsiveness and Oxidative Stress in Animals with Lung Inflammation
  31. Effects of Repeated Stress on Distal Airway Inflammation, Remodeling and Mechanics in an Animal Model of Chronic Airway Inflammation
  32. Effects of inducible nitric oxide synthase inhibition in bronchial vascular remodeling-induced by chronic allergic pulmonary inflammation
  33. Inactivation of capsaicin-sensitive nerves reduces pulmonary remodeling in guinea pigs with chronic allergic pulmonary inflammation
  34. Repeated stress reduces mucociliary clearance in animals with chronic allergic airway inflammation
  35. Effects of pneumonectomy on nitric oxide synthase expression and perivascular edema in the remaining lung of rats
  36. Effects of iNOS Inhibition on Lung Tissue Mechanics and 8-Iso-PGF2α Expression Induced by Physical Stress in Animals with Chronic Pulmonary Inflammation.
  37. Repeated Physical Stress Increases Distal Lung Collagen Remodeling, Eosinophilic Inflammation and iNOS Activation Induced by Chronic Pulmonary Inflammation: Effects of iNOS Inhibition.
  38. Modulation of Distal Lung Parenchyma Mechanics in Guinea Pigs with Chronic Pulmonary Inflammation Submitted to Repeated Physical Stress: Evaluation of Th2 Cytokines Expression.
  39. Inducible nitric oxide synthase inhibition attenuates lung tissue responsiveness and remodeling in a model of chronic pulmonary inflammation in guinea pigs
  40. Oral tolerance attenuates airway inflammation and remodeling in a model of chronic pulmonary allergic inflammation
  41. Effects of chronic l-NAME treatment lung tissue mechanics, eosinophilic and extracellular matrix responses induced by chronic pulmonary inflammation
  42. Oral tolerance attenuates changes in in vitro lung tissue mechanics and extracellular matrix remodeling induced by chronic allergic inflammation in guinea pigs
  43. Capsaicin-sensitive nerves and neurokinins modulate non-neuronal nNOS expression in lung
  44. Effects of Stress and Neuropeptides on Airway Responses in Ovalbumin-Sensitized Rats
  45. Effects of Nitric Oxide Synthases in Chronic Allergic Airway Inflammation and Remodeling
  46. Comparison of early and late responses to antigen of sensitized guinea pig parenchymal lung strips
  47. Effects of acute and chronic nitric oxide inhibition in an experimental model of chronic pulmonary allergic inflammation in guinea pigs
  48. Neurokinins and inflammatory cell iNOS expression in guinea pigs with chronic allergic airway inflammation
  49. Comparison of glucocorticoid and cysteinyl leukotriene receptor antagonist treatments in an experimental model of chronic airway inflammation in guinea-pigs