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  1. Author Correction: Postprandial lipid metabolism durably enhances T cell immunity
  2. Postprandial lipid metabolism durably enhances T cell immunity
  3. Redox and detox: Malate shuttle metabolism keeps exhausted T cells fit
  4. Data from PPAR-Induced Fatty Acid Oxidation in T Cells Increases the Number of Tumor-Reactive CD8<sup>+</sup> T Cells and Facilitates Anti–PD-1 Therapy
  5. Supplementary Table and Figures from PPAR-Induced Fatty Acid Oxidation in T Cells Increases the Number of Tumor-Reactive CD8<sup>+</sup> T Cells and Facilitates Anti–PD-1 Therapy
  6. Supplementary Table and Figures from PPAR-Induced Fatty Acid Oxidation in T Cells Increases the Number of Tumor-Reactive CD8<sup>+</sup> T Cells and Facilitates Anti–PD-1 Therapy
  7. Data from PPAR-Induced Fatty Acid Oxidation in T Cells Increases the Number of Tumor-Reactive CD8<sup>+</sup> T Cells and Facilitates Anti–PD-1 Therapy
  8. Metabolic reprogramming via an engineered PGC-1α improves human chimeric antigen receptor T-cell therapy against solid tumors
  9. Fully murine CD105-targeted CAR-T cells provide an immunocompetent model for CAR-T cell biology
  10. Bionanotechnology in Cancer
  11. Trending approaches in electrospinning and electrospraying for biomedical applications
  12. Tumors attenuating the mitochondrial activity in T cells escape from PD-1 blockade therapy
  13. Targeted suppression of metastasis regulatory transcription factor SOX2 in various cancer cell lines using a sequence-specific designer pyrrole–imidazole polyamide
  14. Nanomedicine: Diagnosis, Treatment, and Potential Prospects
  15. Aptamer Technology for the Detection of Foodborne Pathogens and Toxins
  16. Aptamer-Mediated Nanobiosensing for Health Monitoring
  17. A Combination of Linalool, Vitamin C, and Copper Synergistically Triggers Reactive Oxygen Species and DNA Damage and Inhibits Salmonella enterica subsp. enterica Serovar Typhi and Vibrio fluvialis
  18. PPAR-Induced Fatty Acid Oxidation in T Cells Increases the Number of Tumor-Reactive CD8+ T Cells and Facilitates Anti–PD-1 Therapy
  19. Mitochondrial activation chemicals synergize with surface receptor PD-1 blockade for T cell-dependent antitumor activity