What is it about?
The oxygen-induced oxidative stress is able to modify the cellular profile during inflammation in experimental model of asthma.
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Why is it important?
We have shown for the first time that medical oxygen intensifies the Th17 cell-mediated immune response during experimental model of asthma.
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This page is a summary of: Hyperoxia promotes polarization of the immune response in ovalbumin‐induced airway inflammation, leading to a TH
17
cell phenotype, Immunity Inflammation and Disease, June 2015, Wiley,
DOI: 10.1002/iid3.71.
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