What is it about?
The oxygen-induced oxidative stress is able to modify the cellular profile during inflammation in experimental model of asthma.
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Why is it important?
We have shown for the first time that medical oxygen intensifies the Th17 cell-mediated immune response during experimental model of asthma.
Perspectives
Our expectation is to understand the relationship between oxygen-induced oxidative stress and the Th17 cell-mediated immune response in the pathophysiology of experimental asthma. We hope in the future that this knowledge will contribute with other researchers who aim to control the asthma inflammatory response in humans.
Akinori Cardozo Nagato
Federal University of Juiz de Fora
Read the Original
This page is a summary of: Hyperoxia promotes polarization of the immune response in ovalbumin‐induced airway inflammation, leading to a TH
17
cell phenotype, Immunity Inflammation and Disease, June 2015, Wiley,
DOI: 10.1002/iid3.71.
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